Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Kari A. Hafner"'
Autor:
Lisa A Boardman, Ruth A Johnson, Kimberly B Viker, Kari A Hafner, Robert B Jenkins, Douglas L Riegert-Johnson, Thomas C Smyrk, Kristin Litzelman, Songwon Seo, Ronald E Gangnon, Corinne D Engelman, David N Rider, Russell J Vanderboom, Stephen N Thibodeau, Gloria M Petersen, Halcyon G Skinner
Publikováno v:
PLoS ONE, Vol 8, Iss 11, p e80015 (2013)
IntroductionColorectal cancer (CRC) tumor DNA is characterized by chromosomal damage termed chromosomal instability (CIN) and excessively shortened telomeres. Up to 80% of CRC is microsatellite stable (MSS) and is historically considered to be chromo
Externí odkaz:
https://doaj.org/article/0cbf7004f69043d4a6e8c2961198119c
Publikováno v:
Journal of Biological Chemistry. 279:9389-9391
The size of the CAG tract at the Huntington's disease (HD) locus upon transmission depends on the gender of the parent. However, the basis for the parent-of-origin effect is unknown. To test whether expansion and contraction in HD are "imprinted" in
Autor:
James N. Ingle, Kari A. Hafner, Jennifer R. Ayers-Ringler, Franca Stivala, Evanthia Galanis, Cosima Quatraro, Alexey A. Leontovich, Jeffrey L. Salisbury, Robert B. Jenkins, Massimo Libra, Angela Amato, Antonino B. D'Assoro
Publikováno v:
Europe PubMed Central
Development of chromosomal instability (CIN) and consequent phenotypic heterogeneity represent common events during breast cancer progression. Breast carcinomas harboring extensive chromosomal aberrations display a more aggressive behavior characteri
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d9d4a01fd5709fa37c5957406fba9f31
http://hdl.handle.net/20.500.11769/44143
http://hdl.handle.net/20.500.11769/44143
Prostate cancer cells use genetic and epigenetic mechanisms for progression to androgen independence
Autor:
Robert B. Jenkins, Kari A. Hafner, George Vasmatzis, Donald J. Tindall, Lucy J. Schmidt, Horacio Murillo, Karla V. Ballman, Melissa Karter, Yasushi Kondo
Publikováno v:
Genes, chromosomescancer. 45(7)
Studies on the genetic basis of prostate cancer (PCa) have lead to mixed results with the only consensus being that PCa is a complex disease. Our goal was to gain insight into potential events involved in the acquisition of the androgen-refractory ph
Autor:
Jeffrey M. Slezak, Lynn C. Hartmann, Fergus J. Couch, Colleen S. Sinclair, Thomas A. Sellers, Robert B. Jenkins, Camilo Adem, Kari A. Hafner, Carol Reynolds, Cheryl L. Soderberg, Daniel J. Schaid
Publikováno v:
Genes, chromosomescancer. 41(1)
Breast cancer risk is greatly increased in women who carry mutations in the BRCA1 or BRCA2 genes. Because breast cancer initiation is different between BRCA1/2 mutation carriers and women who do not carry mutations, it is possible that the mechanism
Autor:
Thomas C. Smyrk, Halcyon G. Skinner, Gloria M. Petersen, Ronald E. Gangnon, David N. Rider, Ruth A. Johnson, Kristin Litzelman, Lisa A. Boardman, Kimberly Viker, Stephen N. Thibodeau, Robert B. Jenkins, Kari A. Hafner, Douglas L. Riegert-Johnson, Russell Vanderboom, Songwon Seo, Corinne D. Engelman
Publikováno v:
PLoS ONE
PLoS ONE, Vol 8, Iss 11, p e80015 (2013)
PLoS ONE, Vol 8, Iss 11, p e80015 (2013)
IntroductionColorectal cancer (CRC) tumor DNA is characterized by chromosomal damage termed chromosomal instability (CIN) and excessively shortened telomeres. Up to 80% of CRC is microsatellite stable (MSS) and is historically considered to be chromo