Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Karen L. Hutchinson"'
Autor:
Francois Villinger, Karen L. Hutchinson, Soumya Nidtha, Laure Y. Juompan, David C. Montefiori, Francis J. Novembre
Publikováno v:
AIDS Research and Human Retroviruses. 24:573-586
The mechanisms of resistance to AIDS development in HIV-1-infected chimpanzees have remained elusive. Unique among chimpanzees naturally or experimentally infected with HIV, several animals of the Yerkes cohort have progressed to clinical AIDS with s
Autor:
Pierre E. Rollin, Francois Villinger, Clarence J. Peters, Mary Elizabeth Miranda, Karen L. Hutchinson, Thomas G. Ksiazek
Publikováno v:
Journal of Medical Virology. 65:561-566
Ebola virus (EBO) causes the most severe form of viral hemorrhagic fever in humans and nonhuman primates with up to 90% of infections culminating in death. The requirement of maximum containment laboratories for Ebola virus research has limited oppor
Autor:
Clarence J. Peters, Sherif R. Zaki, Pierre E. Rollin, Karen L. Hutchinson, Patricia W. Greer, Wun-Ju Shieh
Publikováno v:
Journal of Medical Virology. 60:70-76
Black Creek Canal (BCC) virus is a hantavirus associated with hantavirus pulmonary syndrome in southeastern North America. The virus was isolated from the spleen of a cotton rat (Sigmodon hispidus) trapped in southern Florida. Our previous studies ha
Publikováno v:
The American Journal of Tropical Medicine and Hygiene. 59:58-65
This report describes the first detailed analysis of the replication, persistence, and excretion of a North American hantavirus in its natural rodent reservoir. Black Creek Canal virus was isolated from Sigmodon hispidus (cotton rat) shortly after th
Publikováno v:
Virology. 224(1):139-149
Sin Nombre (SN) virus is the major etiologic agent of hantavirus pulmonary syndrome, a severe respiratory disease with high mortality. Like other hantaviruses, SN virus causes an inapparent chronic infection of the natural rodent reservoir and tends
Publikováno v:
Virology. 174:444-449
tsG16(l), a temperature-sensitive mutant of vesicular stomatitis virus, in vitro has at least three phenotypic differences from its parental wild-type (wt) virus due to mutation of the L gene. It was not known whether (i) the temperature-sensitivity
Autor:
Pierre E. Rollin, Michael L. McRae, Karen L. Hutchinson, Bali Pulendran, Sudhanshu Agarwal, Siddhartha Mahanty
Publikováno v:
Scopus-Elsevier
Acute infection of humans with Ebola and Lassa viruses, two principal etiologic agents of hemorrhagic fevers, often results in a paradoxical pattern of immune responses: early infection, characterized by an outpouring of inflammatory mediators such a
Externí odkaz:
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http://www.scopus.com/inward/record.url?eid=2-s2.0-0037443448&partnerID=MN8TOARS