Zobrazeno 1 - 10
of 254
pro vyhledávání: '"Kao, Race"'
Autor:
Zheng, Zhibo, Ma, He, Zhang, Xia, Tu, Fei, Wang, Xiaohui, Ha, Tuanzhu, Fan, Min, Liu, Li, Xu, Jingjing, Yu, Kaijiang, Wang, Ruitao, Kalbfleisch, John, Kao, Race, Williams, David, Li, Chuanfu
Publikováno v:
The Journal of Infectious Diseases, 2017 May 01. 215(9), 1396-1406.
Externí odkaz:
https://www.jstor.org/stable/26364975
Autor:
Ma, He, Wang, Xiaohui, Ha, Tuanzhu, Gao, Ming, Liu, Li, Wang, Ruitao, Yu, Kaijiang, Kalbfleisch, John H., Kao, Race L., Williams, David L., Li, Chuanfu
Publikováno v:
The Journal of Infectious Diseases, 2016 Dec 01. 214(11), 1773-1783.
Externí odkaz:
https://www.jstor.org/stable/26166744
Autor:
Lu, Chen, Wang, Xiaohui, Ha, Tuanzhu, Hu, Yuanping, Liu, Li, Zhang, Xia, Yu, Honghui, Miao, Jonathan, Kao, Race, Kalbfleisch, John, Williams, David, Li, Chuanfu
Publikováno v:
In Journal of Molecular and Cellular Cardiology December 2015 89 Part A:87-97
Autor:
Li, Chuanfu, Hua, Fang, Ha, Tuanzhu, Singh, Krishna, Lu, Chen, Kalbfleisch, John, Breuel, Kevin F., Ford, Tiffany, Kao, Race L., Gao, Ming, Ozment, Tammy R., Williams, David L.
Publikováno v:
ETSU Faculty Works.
Phosphoinositide-3-kinase (PI3K)/Akt dependent signaling has been shown to improve outcome in sepsis/septic shock. There is also ample evidence that PI3K/Akt dependent signaling plays a crucial role in maintaining normal cardiac function. We hypothes
Autor:
Wang, Xiaohui, Ha, Tuanzhu, Liu, Li, Hu, Yuanping, Kao, Race, Kalbfleisch, John, Williams, David, Li, Chuanfu
Publikováno v:
ETSU Faculty Works.
The present study investigated whether TLR3 is required for neonatal heart repair and regeneration following myocardial infarction (MI). TLR3 deficient neonatal mice exhibited impaired cardiac functional recovery and a larger infarct size, while wild
Autor:
Wang, Xiaohui, Ha, Tuanzhu, Hu, Yuanping, Lu, Chen, Liu, Li, Zhang, Xia, Kao, Race, Kalbfleisch, John, Williams, David, Li, Chuanfu
Publikováno v:
ETSU Faculty Works.
Background: Myocardial apoptosis plays an important role in myocardial ischemia/reperfusion (I/R) injury. Activation of PI3K/Akt signaling protects the myocardium from I/R injury. This study investigated the role of miR-214 in hypoxia/ reoxygenation
Autor:
Zhang, Ling, Wang, Hao, Zhao, Yu, Wang, Jianguo, Dubielecka, Patrycja M., Zhuang, Shougang, Qin, Gangjian, Chin, Y. Eugene, Kao, Race L., Zhao, Ting C.
Publikováno v:
ETSU Faculty Works.
Background: Histone deacetylases (HDACs) play a critical role in modulating myocardial protection and cardiomyocyte survivals. However, Specific HDAC isoforms in mediating myocardial ischemia/reperfusion injury remain currently unknown. We used cardi
Autor:
Kao, Race L.
Publikováno v:
ETSU Faculty Works.
Externí odkaz:
https://dc.etsu.edu/etsu-works/19236
Publikováno v:
ETSU Faculty Works.
We have demonstrated that in vitro brief ischemia activates nuclear factor (NF)-κB in rat myocardium. We report in vivo ischemia-reperfusion (I/R)-induced NF-κB activation, IκB kinase -β (IKKβ) activity, and IκBα phosphorylation and degradatio
Externí odkaz:
https://dc.etsu.edu/etsu-works/19155
Autor:
Li, Chuanfu, Ha, Tuanzhu, Kelley, Jim, Gao, Xiang, Qiu, Yufeng, Kao, Race L., Browder, William, Williams, David L.
Publikováno v:
ETSU Faculty Works.
Objective: Immune and inflammatory signaling pathways, initiated by the innate response, are involved in myocardial ischemia/reperfusion (I/R) injury. Toll-like receptor (TLR) mediated MyD88-dependent NFκB pathways play a role in the induction of in
Externí odkaz:
https://dc.etsu.edu/etsu-works/18719