Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Kanae Sakakibara"'
Autor:
Takako Nakahara, Mamoru Ouchida, Akira Kitanaka, Kaoru Tohyama, Kanae Sakakibara, Shin ichiro Suemori, Nami Kurozumi, Takayuki Tsujioka, Masaki Takeuchi, Misako Shibakura
Publikováno v:
Cancer Science
A novel proteasome deubiquitinase inhibitor, VLX1570, has been highlighted as a promising therapeutic agent mainly for lymphoid neoplasms and solid tumors. We examined in vitro effects of VLX1570 on eight myeloid and three lymphoid leukemia cell line
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::10d8b084c9f667de42a169bde4c0f818
https://doi.org/10.1111/cas.14982
https://doi.org/10.1111/cas.14982
Autor:
Takayuki Tsujioka, Kaoru Tohyama, Nami Kurozumi, Shin ichiro Suemori, Akira Kitanaka, Kanae Sakakibara, Yujiro Arao, Jun ichiro Kida, Takako Nakahara
Publikováno v:
International journal of hematology. 110(2)
A MEK1/2 inhibitor, binimetinib is promising as a therapeutic agent for malignant melanoma with N-RAS mutation. We examined in vitro effects of binimetinib on 10 human myeloid/lymphoid leukemia cell lines, and found that three of five cell lines with
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e9070ba005d82c58078222669dd0534c
https://pubmed.ncbi.nlm.nih.gov/31129802
https://pubmed.ncbi.nlm.nih.gov/31129802
Autor:
Shinichiro Suemori, Kanae Sakakibara, Takayuki Tsujioka, Takayuki Takahata, Yumi Tohyama, Jun-ichiro Kida, Kaoru Tohyama, Shuichiro Okamoto, Takahiro Yamauchi, Akira Kitanaka
Publikováno v:
Leukemia. 32(8)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::a17dabad8d68f76ad2d7a86441a46efc
https://pubmed.ncbi.nlm.nih.gov/29955132
https://pubmed.ncbi.nlm.nih.gov/29955132
Autor:
Takayuki Tsujioka, Takahiro Yamauchi, Shinichiro Suemori, Kaoru Tohyama, Kanae Sakakibara, Shuichiro Okamoto, Akira Kitanaka, Jun-ichiro Kida, Yumi Tohyama
Publikováno v:
Blood. 132:5501-5501
Myelodysplastic syndromes (MDS) have a risk of progression to acute myeloid leukemia (AML), but the deterioration mechanisms of MDS and the alteration points still remain to be elucidated. We previously established a myelodysplastic cell line, MDS92
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e9bd42bded92d81787309322ed00d77a
https://doi.org/10.1182/blood-2018-99-110583
https://doi.org/10.1182/blood-2018-99-110583
Autor:
Takehiro Matsubara, Kanae Sakakibara, Hisashi Ishida, Akira Shimada, Kiichiro Kanamitsu, Kana Washio, Hirotaka Kanzaki, Michiko Muraoka
Publikováno v:
Pediatric Blood & Cancer. 63:2059-2060
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::97bb12af35d4eea8c55e5f1eb9077752
https://doi.org/10.1002/pbc.26100
https://doi.org/10.1002/pbc.26100
Autor:
Kanae, Sakakibara, Yoshiki, Okayama, Kenji, Fukushima, Shunsaku, Kaji, Michiko, Muraoka, Yujiro, Arao, Akira, Shimada
Publikováno v:
Pediatrics international : official journal of the Japan Pediatric Society. 57(5)
Congenital factor VII (FVII) deficiency is a rare bleeding disorder with autosomal recessive inheritance. The present female patient was diagnosed with congenital FVII deficiency because of low hepaplastin test (HPT), although vitamin K was given. He
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::2d9c4b4393a8d3982961d33c9c65627d
https://pubmed.ncbi.nlm.nih.gov/26310716
https://pubmed.ncbi.nlm.nih.gov/26310716
Autor:
Shunsaku Kaji, Yoshiki Okayama, Akira Shimada, Kanae Sakakibara, Kenji Fukushima, Michiko Muraoka, Yujiro Arao
Publikováno v:
Pediatrics International. 57:1023-1024
Congenital factor VII (FVII) deficiency is a rare bleeding disorder with autosomal recessive inheritance. The present female patient was diagnosed with congenital FVII deficiency because of low hepaplastin test (HPT), although vitamin K was given. He
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0f328683a187d968a8ec72c46748c8d2
https://doi.org/10.1111/ped.12696
https://doi.org/10.1111/ped.12696