Zobrazeno 1 - 10
of 15
pro vyhledávání: '"Kamila A Marzec"'
Autor:
Alvaro Gonzalez Rajal, Kamila A Marzec, Rachael A McCloy, Max Nobis, Venessa Chin, Jordan F Hastings, Kaitao Lai, Marina Kennerson, William E Hughes, Vijesh Vaghjiani, Paul Timpson, Jason E Cain, D Neil Watkins, David R Croucher, Andrew Burgess
Publikováno v:
eLife, Vol 10 (2021)
We previously used a pulse-based in vitro assay to unveil targetable signalling pathways associated with innate cisplatin resistance in lung adenocarcinoma (Hastings et al., 2020). Here, we advanced this model system and identified a non-genetic mech
Externí odkaz:
https://doaj.org/article/2218265a2a26450eb03d27ef3e339032
Autor:
Ramesh K. Narayanan, Gonzalo Perez-siles, Kamila A. Marzec, Alexandra Boyling, Brent Neumann, Manoj P. Menezes, Marina L. Kennerson
Publikováno v:
Genes and Diseases, Vol 11, Iss 4, Pp 101071- (2024)
Externí odkaz:
https://doaj.org/article/1a0652a16abf43909617a03e75b833d3
Autor:
Kamila A. Marzec, Samuel Rogers, Rachael McCloy, Benjamin L. Parker, David E. James, D. Neil Watkins, Andrew Burgess
Publikováno v:
Scientific Reports, Vol 12, Iss 1, Pp 1-13 (2022)
Abstract Microtubule-associated serine/threonine kinase-like (MASTL) has emerged as a critical regulator of mitosis and as a potential oncogene in a variety of cancer types. To date, Arpp-19/ENSA are the only known substrates of MASTL. However, with
Externí odkaz:
https://doaj.org/article/3331cc1a3f214642bc1ecc88d07abe93
Autor:
Kamila A Marzec, Samuel Rogers, Rachael McCloy, Benjamin L Parker, David E James, D. Neil Watkins, Andrew Burgess
MASTL (microtubule-associated serine/threonine kinase-like) has emerged as a critical regulator of mitosis and as a potential oncogene in a variety of cancer types. To date, Arpp-19/ENSA are the only known substrates of MASTL. However, with the roles
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::86c09b272eb026e0373aee9ad7de2588
https://doi.org/10.21203/rs.3.rs-1503557/v1
https://doi.org/10.21203/rs.3.rs-1503557/v1
Autor:
Alvaro Gonzalez Rajal, Andrew Burgess, Rachael A. McCloy, William E. Hughes, D. Neil Watkins, Paul Timpson, Kaitao Lai, Max Nobis, Marina L. Kennerson, Jason E. Cain, Vijesh Vaghjiani, Venessa T. Chin, Jordan F. Hastings, David R. Croucher, Kamila A Marzec
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::d8e4f3c94d6f61e02d92a9b65ba05572
https://doi.org/10.7554/elife.65234.sa2
https://doi.org/10.7554/elife.65234.sa2
Autor:
Paul Timpson, Rachael A. McCloy, William E. Hughes, Andrew Burgess, Max Nobis, Kamila A Marzec, Vijesh Vaghjiani, Venessa T. Chin, Marina L. Kennerson, Jason E. Cain, Alvaro Gonzalez Rajal, Jordan F. Hastings, David R. Croucher, D. Neil Watkins, Kaitao Lai
Publikováno v:
eLife
eLife, Vol 10 (2021)
eLife, Vol 10 (2021)
We previously used a pulse-based in vitro assay to unveil targetable signalling pathways associated with innate cisplatin resistance in lung adenocarcinoma (Hastings et al., 2020). Here, we advanced this model system and identified a non-genetic mech
Autor:
Lars Juhl Jensen, Ulrik Nicolai de Lichtenberg, Jenny Vuong, Andrew Burgess, Kamila A. Marzec, Seán I. O'Donoghue
Publikováno v:
Cell. 179(3)
S-phase entry and exit are regulated by hundreds of protein complexes that assemble “just in time,” orchestrated by a multitude of distinct events. To help understand their interplay, we have created a tailored visualization based on the Minardo
Publikováno v:
Cancer Letters. 381:149-155
We previously showed that BARD1 is a shuttling protein with pro-apoptotic activity in MCF-7 breast cancer cells. BARD1 is expressed as splice variant isoforms in breast cancer. Here we characterized YFP-tagged BARD1 splice variants (beta, omega, phi,
Publikováno v:
Oncotarget
Chemotherapy drugs that induce apoptosis by causing DNA double-strand breaks, upregulate the tumor suppressor p53. This study investigated the regulation of the growth-regulatory protein insulin-like growth factor binding protein-3 (IGFBP-3), a p53 t
Publikováno v:
BioMed Research International
BioMed Research International, Vol 2015 (2015)
BioMed Research International, Vol 2015 (2015)
Insulin-like growth factor binding protein-3 (IGFBP-3) is a key regulatory molecule of the IGF axis and can function in a tissue-specific way as both a tumor suppressor and promoter. Triple-negative breast cancer (TNBC) has high tumor expression of I