Zobrazeno 1 - 10
of 21
pro vyhledávání: '"Kalina Andrysiak"'
Autor:
Izabela Kraszewska, Mateusz Tomczyk, Kalina Andrysiak, Monika Biniecka, Anja Geisler, Henry Fechner, Michał Zembala, Jacek Stępniewski, Józef Dulak, Agnieszka Jaźwa-Kusior
Publikováno v:
Molecular Therapy: Methods & Clinical Development, Vol 17, Iss , Pp 1190-1201 (2020)
Systemically delivered adeno-associated viral vector serotype 9 (AAV9) effectively transduces murine heart, but provides transgene expression also in liver and skeletal muscles. Improvement of the selectivity of transgene expression can be achieved t
Externí odkaz:
https://doaj.org/article/e31943af4dc24b31b50d9af45a05ad0e
Autor:
Kalina Andrysiak, Alicja Martyniak, Anna Potulska-Chromik, Anna Kostera-Pruszczyk, Jacek Stępniewski, Józef Dulak
Publikováno v:
Stem Cell Research, Vol 57, Iss , Pp 102563- (2021)
Spinal Muscular Atrophy (SMA) is a genetic neuromuscular disease caused by mutations inSMN1 gene encoding survival motor neuron (SMN) protein. Lack of this protein leads to progressive loss of motor neurons and therefore to gradual loss of signal tra
Externí odkaz:
https://doaj.org/article/7bc2e014b8df4854b25bfcb570300c17
Autor:
Paulina Podkalicka, Olga Mucha, Katarzyna Kaziród, Iwona Bronisz-Budzyńska, Sophie Ostrowska-Paton, Mateusz Tomczyk, Kalina Andrysiak, Jacek Stępniewski, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Biomedicines, Vol 9, Iss 5, p 481 (2021)
Duchenne muscular dystrophy (DMD), caused by a lack of functional dystrophin, is characterized by progressive muscle degeneration. Interestingly, dystrophin is also expressed in endothelial cells (ECs), and insufficient angiogenesis has already been
Externí odkaz:
https://doaj.org/article/3c0bb1db19374aebb98eca93a846744d
Autor:
Mateusz Jeż, Alicja Martyniak, Kalina Andrysiak, Olga Mucha, Krzysztof Szade, Alan Kania, Łukasz Chrobok, Katarzyna Palus-Chramiec, Anna M. Sanetra, Marian H. Lewandowski, Ewelina Pośpiech, Jacek Stępniewski, Józef Dulak
Publikováno v:
Cells, Vol 10, Iss 3, p 522 (2021)
Heme oxygenase-1 (HO-1, encoded by HMOX1) is a cytoprotective enzyme degrading heme into CO, Fe2+, and biliverdin. HO-1 was demonstrated to affect cardiac differentiation of murine pluripotent stem cells (PSCs), regulate the metabolism of murine adul
Externí odkaz:
https://doaj.org/article/c6f175a152ef436881f837a7de6be858
Autor:
Jacek Stępniewski, Mateusz Tomczyk, Kalina Andrysiak, Izabela Kraszewska, Alicja Martyniak, Agnieszka Langrzyk, Klaudia Kulik, Ewa Wiśniewska, Mateusz Jeż, Urszula Florczyk-Soluch, Katarzyna Polak, Paulina Podkalicka, Neli Kachamakova-Trojanowska, Alicja Józkowicz, Agnieszka Jaźwa-Kusior, Józef Dulak
Publikováno v:
Biomedicines, Vol 8, Iss 12, p 578 (2020)
Cell therapies are extensively tested to restore heart function after myocardial infarction (MI). Survival of any cell type after intracardiac administration, however, may be limited due to unfavorable conditions of damaged tissue. Therefore, the aim
Externí odkaz:
https://doaj.org/article/4ee98a8a965f46a2a1af27dff3ead409
Autor:
Paulina Podkalicka, Olga Mucha, Szczepan Kruczek, Anna Biela, Kalina Andrysiak, Jacek Stępniewski, Maciej Mikulski, Michał Gałęzowski, Kamil Sitarz, Krzysztof Brzózka, Alicja Józkowicz, Józef Dulak, Agnieszka Łoboda
Publikováno v:
Biomolecules, Vol 10, Iss 1, p 143 (2020)
Elevated expression of heme oxygenase-1 (HO-1, encoded by HMOX1) is observed in various types of tumors. Hence, it is suggested that HO-1 may serve as a potential target in anticancer therapies. A novel approach to inhibit HO-1 is related to the synt
Externí odkaz:
https://doaj.org/article/abc43edecb304adeb1ffd2bee44f409e
Autor:
Urszula Głowniak-Kwitek, Asier Laria Caballero, Iwona Bronisz-Budzyńska, Magdalena Kozakowska, Kalina Andrysiak, Jacek Stępniewski, Agnieszka Łoboda, Józef Dulak
Publikováno v:
Pharmacological Reports. 75:397-410
Background Impaired muscle regeneration is a hallmark of Duchenne muscular dystrophy (DMD), a neuromuscular disorder caused by mutations in the DMD gene encoding dystrophin. The lack of heme oxygenase-1 (HO-1, Hmox1), a known anti-inflammatory and cy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7532224e3ab3185a4018117459f8d677
https://doi.org/10.1007/s43440-023-00475-3
https://doi.org/10.1007/s43440-023-00475-3
Autor:
Kalina Andrysiak, Jacek Stępniewski, Michał Zembala, Monika Biniecka, Izabela Kraszewska, Henry Fechner, Agnieszka Jaźwa-Kusior, Jozef Dulak, Mateusz Tomczyk, Anja Geisler
Publikováno v:
Molecular Therapy: Methods & Clinical Development, Vol 17, Iss, Pp 1190-1201 (2020)
Molecular Therapy. Methods & Clinical Development
Molecular Therapy. Methods & Clinical Development
Systemically delivered adeno-associated viral vector serotype 9 (AAV9) effectively transduces murine heart, but provides transgene expression also in liver and skeletal muscles. Improvement of the selectivity of transgene expression can be achieved t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5bd5171e1f34586331775bb649ff9bab
https://doi.org/10.1016/j.omtm.2020.05.006
https://doi.org/10.1016/j.omtm.2020.05.006
Autor:
Alicja Martyniak, Jozef Dulak, Jacek Stępniewski, Anna Potulska-Chromik, Kalina Andrysiak, Anna Kostera-Pruszczyk
Publikováno v:
Stem Cell Research, Vol 57, Iss, Pp 102563-(2021)
Spinal Muscular Atrophy (SMA) is a genetic neuromuscular disease caused by mutations inSMN1 gene encoding survival motor neuron (SMN) protein. Lack of this protein leads to progressive loss of motor neurons and therefore to gradual loss of signal tra
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::74aaa8afb75582ffd52750209aeb7d35
https://pubmed.ncbi.nlm.nih.gov/34678665
https://pubmed.ncbi.nlm.nih.gov/34678665
Autor:
Jacek Stępniewski, Paulina Koczurkiewicz-Adamczyk, Kalina Andrysiak, Sławomir Lasota, Katarzyna Wójcik-Pszczoła, Agnieszka Gunia-Krzyżak, Jozef Dulak, Kamil Piska, Katarzyna Klaś, Zbigniew Madeja, Elżbieta Pękala
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 6217, p 6217 (2021)
International Journal of Molecular Sciences
Volume 22
Issue 12
International Journal of Molecular Sciences
Volume 22
Issue 12
Doxorubicin (DOX) is a widely used anticancer drug. However, its clinical use is severely limited due to drug-induced cumulative cardiotoxicity, which leads to progressive cardiomyocyte dysfunction and heart failure. Enormous efforts have been made t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::879c9cd2061ccbd93b4fc4555f4ad8e4
https://www.mdpi.com/1422-0067/22/12/6217
https://www.mdpi.com/1422-0067/22/12/6217