Zobrazeno 1 - 10
of 16
pro vyhledávání: '"Kaida Huang"'
Autor:
Yubiao Lin, Kaida Huang, Zhezhen Cai, Yide Chen, Lihua Feng, Yingqin Gao, Wenhui Zheng, Xin Fan, Guoqin Qiu, Jianmin Zhuang, Shuitu Feng
Publikováno v:
Frontiers in Pharmacology, Vol 13 (2022)
Objective: Gastric cancer (GC) is a highly heterogeneous malignant carcinoma. This study aimed to conduct an exosome-based classification for assisting personalized therapy for GC.Methods: Based on the expression profiling of prognostic exosome-relat
Externí odkaz:
https://doaj.org/article/b5d84edce54d4d9e98dc7b514b7ff26e
Publikováno v:
Journal of Hepatocellular Carcinoma.
Kaida Huang,1 Yubiao Lin,1 Keyin Wang,2 Jianfen Shen,3 Dahai Wei2â 4 1Department of Oncology, Xiamen Haicang Hospital, Xiamen, Peopleâs Republic of China; 2Department of Infectious Diseases, Affiliated Hospital of Jiaxing University, Jiaxin
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5bc8fc31da11cc2457abb98b2bb3bacf
https://www.dovepress.com/arfip2-regulates-emt-and-autophagy-in-hepatocellular-carcinoma-in-part-peer-reviewed-fulltext-article-JHC
https://www.dovepress.com/arfip2-regulates-emt-and-autophagy-in-hepatocellular-carcinoma-in-part-peer-reviewed-fulltext-article-JHC
Publikováno v:
Biochemical and Biophysical Research Communications.
Osteoporosis is a prevalent metabolic bone disease with low bone mass and degenerative disorders of bone tissues. Abnormal osteogenic differentiation of bone marrow mesenchymal stem cells (MSCs) is involved in osteoporosis progression. Nevertheless,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::339a936c3691a47c0e1495a5f913571a
https://doi.org/10.1016/j.bbrc.2021.08.045
https://doi.org/10.1016/j.bbrc.2021.08.045
Publikováno v:
Oncology Letters. May2016, Vol. 11 Issue 5, p3477-3482. 6p.
Publikováno v:
International Immunopharmacology. 92:107304
Regenerating islet-derived protein 1-alpha (REG1A) was abnormally upregulated in a series of gastrointestinal inflammatory disorders. However, the potential biological function and underlying regulatory mechanisms of the increased REG1A in inflammato
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::6ad70959b26d24d6ce9b3ae4b12179dd
https://doi.org/10.1016/j.intimp.2020.107304
https://doi.org/10.1016/j.intimp.2020.107304
Autor:
Shihai Wang, Cailin Liu, Liang Ming, Nan Li, Huijun Ren, Min Xu, Xiaogai Li, Junfen Ma, Kaida Huang, Xiaoxu Sun
Publikováno v:
Cancer Biology & Therapy. 17:684-692
Transmembrane tumor necrosis factor-α (tmTNF-α) is known to induce the activation of NF-κB to protect tumor cells. Upregulation of tmTNF-α leads to resistance to apoptosis and induces drug resistance in breast cancer. However, the expression of t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::67d7a5b83c5c63d7fa4c93c4b4bceba7
https://doi.org/10.1080/15384047.2016.1187551
https://doi.org/10.1080/15384047.2016.1187551
Publikováno v:
Bioscience Reports; Jan2021, Vol. 41 Issue 1, p1-12, 12p
Publikováno v:
AIP Conference Proceedings; 2019, Vol. 2073 Issue 1, p020051-1-020051-9, 9p
Publikováno v:
Oncology letters. 11(5)
The aim of the present study was to evaluate the effects of thymidylate synthase (TYMS) and excision repair cross-complementing 1 (ERCC1) polymorphisms on chemotherapeutic efficacy in patients with gastrointestinal tumors using peripheral venous bloo
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d7ed0eadc98eb8b241c8580f6240e83a
https://pubmed.ncbi.nlm.nih.gov/27123139
https://pubmed.ncbi.nlm.nih.gov/27123139
Publikováno v:
Molecular medicine reports. 12(4)
Liver transplantation is the standard treatment for end‑stage liver failure; however, rejection can result in allograft failure. In order to investigate the role of Notch 1 during rejection, the present study evaluated Notch 1 expression, as well a
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::db34b1432b8e1f91b83e55406bd26bde
https://pubmed.ncbi.nlm.nih.gov/26134952
https://pubmed.ncbi.nlm.nih.gov/26134952