Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Kai eSchönig"'
Autor:
Anja eScharinger, Stephanie eEckrich, David H Vandael, Kai eSchönig, Alexandra eKoschak, Dietmar eHecker, Gurjot eKaur, Amy eLee, Anupam eSah, Dusan eBartsch, Bruno eBenedetti, Andreas eLieb, Bernhard eSchick, Nicolas eSingewald, Martina J Brauns, Emilio eCarbone, Jutta eEngel, Joerg eStriessnig
Publikováno v:
Frontiers in Cellular Neuroscience, Vol 9 (2015)
Cav1.3 L-type Ca2+-channel function is regulated by a C-terminal automodulatory domain (CTM). It affects channel binding of calmodulin and thereby tunes channel activity by interfering with Ca2+- and voltage-dependent gating. Alternative splicing gen
Externí odkaz:
https://doaj.org/article/3662552889844fe5b03051f69f53953c
Autor:
Tomas ePetrasek, Iva eProkopova, Martin eSladek, Kamila eWeissova, Iveta eVojtechova, Stepan eBahnik, Anna eZemanova, Kai eSchönig, Stefan eBerger, Björn eTews, Dusan eBartsch, Martin eSchwab, Alena eSumova, Ales eStuchlik
Publikováno v:
Frontiers in Behavioral Neuroscience, Vol 8 (2014)
Decreased levels of Nogo-A dependent signaling have been shown to affect behavior and cognitive functions. In Nogo-A knockout and knock-down laboratory rodents, behavioral alterations were observed, possibly corresponding with human neuropsychiatric
Externí odkaz:
https://doaj.org/article/5502254f725546e3940e84b258dc1b2f
Publikováno v:
Frontiers in Behavioral Neuroscience, Vol 8 (2014)
Nogo-A is an important neurite growth-regulatory protein in the adult and developing nervous system. Mice lacking Nogo-A, or rats with neuronal Nogo-A deficiency, exhibit behavioral abnormalities such as impaired short-term memory, decreased prepulse
Externí odkaz:
https://doaj.org/article/10c0c5dd859348acab25e828d647e10f
Autor:
Zdena eKristofikova, Monika eVrajova, Jana eSirova, Karel eVales, Tomas ePetrasek, Kai eSchönig, Bjorn eTews, Martin eSchwab, Dusan eBartsch, Ales eStuchlik, Daniela eRipova
Publikováno v:
Frontiers in Behavioral Neuroscience, Vol 7 (2013)
It has been suggested that Nogo-A, a myelin-associated protein, could play a role in the pathogenesis of schizophrenia and that Nogo-A-deficient rodents could serve as an animal model for schizophrenic symptoms. Since changes in brain laterality are
Externí odkaz:
https://doaj.org/article/a0565654a0644f3e96063028437c5668
Autor:
Tomas ePetrasek, Iva eProkopova, Martin eSladek, Kamila eWeissova, Iveta eVojtechova, Štěpán eBahník, Anna eZemanova, Kai eSchonig, Stefan eBerger, Bjorn eTews, Dusan eBartsch, Martin E Schwab, Alena eSumova, Ales eStuchlik
Publikováno v:
Frontiers in Behavioral Neuroscience, Vol 8 (2014)
Externí odkaz:
https://doaj.org/article/b601541b9eac492696e9505473d1fd49