Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Kai Wohlfahrt"'
Autor:
Meike Wiese, Andrew J. Bannister, Srinjan Basu, Wayne Boucher, Kai Wohlfahrt, Maria A. Christophorou, Michael L. Nielsen, David Klenerman, Ernest D. Laue, Tony Kouzarides
Publikováno v:
Epigenetics & Chromatin, Vol 12, Iss 1, Pp 1-17 (2019)
Abstract Background Stem cell differentiation involves major chromatin reorganisation, heterochromatin formation and genomic relocalisation of structural proteins, including heterochromatin protein 1 gamma (HP1γ). As the principal reader of the repr
Externí odkaz:
https://doaj.org/article/3abeefb38c8647619e3e0901093a5eca
Publikováno v:
Dermatologic Surgery. 36:2182-2187
BACKGROUND Botulinum toxin (BT) is a safe and effective treatment for cosmetic indications. Formation of BT antibodies can occur but has previously been reported in cosmetic indications in two cases only. OBJECTIVE To report another four patients wit
Autor:
Hans Bigalke, Wendelin Blersch, Oliver Opatz, Arne May, Kai Wohlfahrt, Wilhelm Schulte-Mattler
Publikováno v:
Journal of the Neurological Sciences. 260:38-42
A genuine peripheral antinociceptive and anti-inflammatory effect of Botulinum neurotoxin type A (BoNT/A) has been proposed but could not be demonstrated in humans so far. Therefore, 100 mouse units of Botulinum toxin A (Dysport) and placebo were inj
Publikováno v:
Muscle & Nerve. 15:1138-1142
M responses and twitch contractions were evoked in single motor units (MUs) of the first dorsal interosseus muscle by intramuscular microstimulation of motor axons. Two-hundred nine MUs were studied in 21 subjects. Thirty-five MUs (17%) showed F wave
Autor:
Clemens Allgaier, Kai Wohlfahrt, Antoni Camins, Katia Grüner, Ester Verdaguer, Mercè Pallàs, Florian Wegner, Susana Garcia de Arriba, Armin Wagner
Publikováno v:
Neurochemistry international. 49(5)
In the present study, human NT2 neurons obtained from embryonic teratocarcinoma (NT2) cells were established as human in-vitro model to investigate the mechanisms associated with hypoxia/ischemia-induced neuronal injury. NT2 neurons express functiona