Výsledky vyhledávání - "Kai, Hideaki"

Additional file 5: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded AÎ˛ and prion proteins

Autor: Rossi, Marcello, Kai, Hideaki, Baiardi, Simone, Bartoletti-Stella, Anna, CarlĂ, Benedetta, Zenesini, Corrado, Capellari, Sabina, Tetsuyuki Kitamoto, Parchi, Piero

Table S5. Influence of PRNP codon 129 and PrPSc type on AD pathology. Relative risk ratio (RRR) was calculated by a multinomial logistic regression adjusted for age at death. For independent variables, PRNP genotype homozygous for methionine (MM) and

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Additional file 3: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded AÎ˛ and prion proteins

Autor: Rossi, Marcello, Kai, Hideaki, Baiardi, Simone, Bartoletti-Stella, Anna, CarlĂ, Benedetta, Zenesini, Corrado, Capellari, Sabina, Tetsuyuki Kitamoto, Parchi, Piero

Table S3. Influence of CJD histotype and strain on AD pathology. Relative risk ratio (RRR) was calculated by a multinomial logistic regression adjusted for age at death. For independent variables, MM(V)1 histotype and M1 strain were set as reference

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Additional file 6: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

Autor: Rossi, Marcello, Kai, Hideaki, Baiardi, Simone, Bartoletti-Stella, Anna, Carlà, Benedetta, Zenesini, Corrado, Capellari, Sabina, Tetsuyuki Kitamoto, Parchi, Piero

Table S6. Influence of PRNP mutations on AD pathology. Relative risk ratio (RRR) was calculated by a multinomial logistic regression adjusted for age at death. For independent variables, sCJDMM(V)1 and gCJD E200K were set as reference groups for the

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Additional file 8: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded Aβ and prion proteins

Autor: Rossi, Marcello, Kai, Hideaki, Baiardi, Simone, Bartoletti-Stella, Anna, Carlà, Benedetta, Zenesini, Corrado, Capellari, Sabina, Tetsuyuki Kitamoto, Parchi, Piero

Table S7. Comparison of clinical course between CJD/AD and CJD/notAD groups. The two groups include cases with High or Intermediate (CJD/AD group) and Low or Not (CJD) AD pathological changes, according to the ABC score. *According to Zerr et al. (Ze

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Additional file 2: of The characterization of AD/PART co-pathology in CJD suggests independent pathogenic mechanisms and no cross-seeding between misfolded AÎ˛ and prion proteins

Autor: Rossi, Marcello, Kai, Hideaki, Baiardi, Simone, Bartoletti-Stella, Anna, CarlĂ, Benedetta, Zenesini, Corrado, Capellari, Sabina, Tetsuyuki Kitamoto, Parchi, Piero

Table S2. Correlation analysis between duration of disease and AD/PART pathology. Relative risk ratio (RRR) was calculated by a multinomial logistic regression adjusted by age at death. The lower grades of pathology were chosen as reference categorie

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