Zobrazeno 1 - 4
of 4
pro vyhledávání: '"Kadidia P. Adula"'
Autor:
Kadidia P. Adula, Alvaro Sagasti
Publikováno v:
Methods in Molecular Biology ISBN: 9781071630112
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::6330d0bb90df69a95720a2b619837182
https://doi.org/10.1007/978-1-0716-3012-9_14
https://doi.org/10.1007/978-1-0716-3012-9_14
Autor:
Shigeki Watanabe, Kristina Lippmann, M. Wayne Davis, Erik M. Jorgensen, Sumana Raychaudhuri, Grant F. Kusick, Morven Chin, Kadidia P. Adula, Edward J. Hujber, Thien N Vu
Publikováno v:
Nature neuroscience
Synaptic vesicles fuse with the plasma membrane to release neurotransmitter following an action potential, after which new vesicles must refill vacated release sites. How many vesicles can fuse at a single active zone, where they fuse within the acti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::5eb1e54e96550cacb2ebcacadcb04bd1
https://doi.org/10.1101/509216
https://doi.org/10.1101/509216
Autor:
Patrizia Casaccia, Giulio Maria Pasinetti, Pavan Bhargava, Jimmy L. Huynh, Peter A. Calabresi, Corey A. McGraw, Ilana Katz Sand, Jeffery D. Haines, Merina Varghese, Fred D. Lublin, Aaron E. Miller, Norman J. Haughey, Oscar G. Vidaurre, Weijia Zhang, Elias S. Sotirchos, Veera Venkata Ratnam Bandaru, Fan Zhang, Gang Wu, Kadidia P. Adula, Matilde Inglese
Publikováno v:
Brain. 137:2271-2286
Axonal damage is a prominent cause of disability and yet its pathogenesis is incompletely understood. Using a xenogeneic system, here we define the bioenergetic changes induced in rat neurons by exposure to cerebrospinal fluid samples from patients w
Autor:
Nebojsa Kezunovic, Oscar G. Vidaurre, George W. Huntley, Kadidia P. Adula, Yunjiao Zhu, Maureen Wentling, Patrizia Casaccia
Publikováno v:
The Journal of neuroscience : the official journal of the Society for Neuroscience. 37(31)
Calcium-dependent nuclear export of histone deacetylase 1 (HDAC1) was shown previously to precede axonal damage in culture, but the in vivo relevance of these findings and the potential posttranslational modifications of HDAC1 remained elusive. Using