Zobrazeno 1 - 10
of 20
pro vyhledávání: '"Kadiam C. Venkata Subbaiah"'
Autor:
Omar M. Hedaya, Kadiam C. Venkata Subbaiah, Feng Jiang, Li Huitong Xie, Jiangbin Wu, Eng-Soon Khor, Mingyi Zhu, David H. Mathews, Chris Proschel, Peng Yao
Publikováno v:
Nature Communications, Vol 14, Iss 1, Pp 1-17 (2023)
Abstract Translation of upstream open reading frames (uORFs) typically abrogates translation of main (m)ORFs. The molecular mechanism of uORF regulation in cells is not well understood. Here, we data-mined human and mouse heart ribosome profiling ana
Externí odkaz:
https://doaj.org/article/429d280020be4123b6dae1a44cb7e724
Autor:
Kadiam C. Venkata Subbaiah
Publikováno v:
Cardiogenetics, Vol 13, Iss 1, Pp 31-32 (2023)
Arrhythmogenic cardiomyopathy (ACM) is an inherited multifaceted cardiac disease that causes sudden cardiac death, especially in young adults and athletes [...]
Externí odkaz:
https://doaj.org/article/50da401546574461adf9e565f3de6dcb
Publikováno v:
JCI Insight, Vol 7, Iss 13 (2022)
Cardiac pathological remodeling, a primary contributor to heart failure (HF) and death, is an important target for HF therapy. However, the signaling pathways that govern cardiac remodeling are not fully elucidated. Here, we found that a functionally
Externí odkaz:
https://doaj.org/article/46b585cfe40c49d98cba3e7b49872458
Publikováno v:
Journal of Cardiovascular Development and Disease, Vol 10, Iss 2, p 52 (2023)
Cardiac fibrosis is a primary contributor to heart failure (HF), and is considered to be a targetable process for HF therapy. Cardiac fibroblast (CF) activation accompanied by excessive extracellular matrix (ECM) production is central to the initiati
Externí odkaz:
https://doaj.org/article/5b0c95d169ce4f95bb151fd824e143e1
Publikováno v:
Computational and Structural Biotechnology Journal, Vol 17, Iss , Pp 1326-1338 (2019)
Alteration of RNA structure by environmental signals is a fundamental mechanism of gene regulation. For example, the riboswitch is a noncoding RNA regulatory element that binds a small molecule and causes a structural change in the RNA, thereby regul
Externí odkaz:
https://doaj.org/article/fbeb3a4d4120468ca3312a1b485c3b7f
Autor:
Jiangbin Wu, Kadiam C Venkata Subbaiah, Omar Hedaya, Si Chen, Joshua Munger, Wai Hong Wilson Tang, Chen Yan, Peng Yao
Publikováno v:
bioRxiv
AimsMitochondria play a vital role in cellular metabolism and energetics and support normal cardiac function. Disrupted mitochondrial function and homeostasis cause a variety of heart diseases. Fam210a (family with sequence similarity 210 member A),
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ef0d59d4c7eeca08131782bd967b19d5
https://europepmc.org/articles/PMC10245825/
https://europepmc.org/articles/PMC10245825/
Publikováno v:
JCI insight. 7(13)
Cardiac pathological remodeling, a primary contributor to heart failure (HF) and death, is an important target for HF therapy. However, the signaling pathways that govern cardiac remodeling are not fully elucidated. Here, we found that a functionally
Publikováno v:
Biochem Biophys Res Commun
The GAIT (gamma-interferon-activated inhibitor of translation) complex or miR-297-RISC (RNA-induced silencing complex), together with hnRNP L or hnRNP L-bearing complex, operates an RNA switch in myeloid cells that regulates stress-dependent expressi
Publikováno v:
Computational and Structural Biotechnology Journal, Vol 17, Iss, Pp 1326-1338 (2019)
Computational and Structural Biotechnology Journal
Computational and Structural Biotechnology Journal
Graphical abstract
Alteration of RNA structure by environmental signals is a fundamental mechanism of gene regulation. For example, the riboswitch is a noncoding RNA regulatory element that binds a small molecule and causes a structural change i
Alteration of RNA structure by environmental signals is a fundamental mechanism of gene regulation. For example, the riboswitch is a noncoding RNA regulatory element that binds a small molecule and causes a structural change i
Cardiac fibrosis, a primary contributor to heart failure (HF) and sudden death, is considered as an important target for HF therapy. However, the signaling pathways that govern cardiac fibroblast (CF) function during cardiac fibrosis have not been fu
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::2694f7ba272b8323568555ad91e5563a
https://doi.org/10.1101/2021.02.20.432115
https://doi.org/10.1101/2021.02.20.432115