Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Kacey L. Caradonna"'
Autor:
Yuan Li, Sheena Shah-Simpson, Kwame Okrah, A Trey Belew, Jungmin Choi, Kacey L Caradonna, Prasad Padmanabhan, David M Ndegwa, M Ramzi Temanni, Héctor Corrada Bravo, Najib M El-Sayed, Barbara A Burleigh
Publikováno v:
PLoS Pathogens, Vol 12, Iss 4, p e1005511 (2016)
Intracellular colonization and persistent infection by the kinetoplastid protozoan parasite, Trypanosoma cruzi, underlie the pathogenesis of human Chagas disease. To obtain global insights into the T. cruzi infective process, transcriptome dynamics w
Externí odkaz:
https://doaj.org/article/64b098f26aac404f98716b062479c8bd
Autor:
Crystal Teygong, Torsten Wittmann, Ira J. Blader, Sheena Shah-Simpson, Kacey L. Caradonna, Barbara A. Burleigh, Xiaoyan Zhao, Niels Galjart, Praveen Kumar
Publikováno v:
Cellular Microbiology. 15:571-584
Summary Mammalian cell invasion by the protozoan parasite Trypanosoma cruzi involves host cell microtubule dynamics. Microtubules support kinesin-dependent anterograde trafficking of host lysosomes to the cell periphery where targeted lysosome exocyt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::98e6450431af007c339775d715233f6f
https://doi.org/10.1111/cmi.12056
https://doi.org/10.1111/cmi.12056
Publikováno v:
Infection and Immunity. 79:2112-2119
Trypanosoma cruzi , the protozoan parasite that causes human Chagas' disease, induces a type I interferon (IFN) (IFN-α/β) response during acute experimental infection in mice and in isolated primary cell types. To examine the potential impact of th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::1b1c1f7c66fa7e67a5f5092c60530858
https://doi.org/10.1128/iai.01176-10
https://doi.org/10.1128/iai.01176-10
Autor:
Li Liu, James D. Bangs, Emilia K. Kruzel, Barbara A. Burleigh, Carlos B. Hirschberg, Yu-Xin Xu, Kacey L. Caradonna
Publikováno v:
The Journal of biological chemistry. 288(15)
Nucleotide sugar transporters (NSTs) are indispensible for the biosynthesis of glycoproteins by providing the nucleotide sugars needed for glycosylation in the lumen of the Golgi apparatus. Mutations in NST genes cause human and cattle diseases and i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::33caee5e13aaffd107f746593aeca7b1
https://pubmed.ncbi.nlm.nih.gov/23443657
https://pubmed.ncbi.nlm.nih.gov/23443657
Autor:
Xiaoyan, Zhao, Praveen, Kumar, Sheena, Shah-Simpson, Kacey L, Caradonna, Niels, Galjart, Crystal, Teygong, Ira, Blader, Torsten, Wittmann, Barbara A, Burleigh
Publikováno v:
Cellular microbiology. 15(4)
Mammalian cell invasion by the protozoan parasite Trypanosoma cruzi involves host cell microtubule dynamics. Microtubules support kinesin-dependent anterograde trafficking of host lysosomes to the cell periphery where targeted lysosome exocytosis eli
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::fe1f7c4d21b2ad2dd97805e7c3bfa9fd
https://pubmed.ncbi.nlm.nih.gov/23107073
https://pubmed.ncbi.nlm.nih.gov/23107073
Publikováno v:
Advances in parasitology. 76
One of the more accepted concepts in our understanding of the biology of early Trypanosoma cruzi-host cell interactions is that the mammalian-infective trypomastigote forms of the parasite must transit the host cell lysosomal compartment in order to
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::9095052373361a84e4007bcef155de5c
https://pubmed.ncbi.nlm.nih.gov/21884886
https://pubmed.ncbi.nlm.nih.gov/21884886
One of the more accepted concepts in our understanding of the biology of early Trypanosoma cruzi–host cell interactions is that the mammalian-infective trypomastigote forms of the parasite must transit the host cell lysosomal compartment in order t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::e210cd8cc81ce226a95c61fe92fa9ef6
https://doi.org/10.1016/b978-0-12-385895-5.00002-5
https://doi.org/10.1016/b978-0-12-385895-5.00002-5
Publikováno v:
Infection and immunity. 77(4)
The Chagas’ disease parasiteTrypanosoma cruzicommonly infects humans through skin abrasions or mucosa from reduviid bug excreta. Yet most studies on animal models start with subcutaneous or intraperitoneal injections, a distant approximation of the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c1dc8fe5b01dd1d0393a37df42d4d374
https://pubmed.ncbi.nlm.nih.gov/19168740
https://pubmed.ncbi.nlm.nih.gov/19168740
Publikováno v:
The FASEB Journal. 22
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f40d6fb3553212da618f1fee050eba28
https://doi.org/10.1096/fasebj.22.1_supplement.707.21
https://doi.org/10.1096/fasebj.22.1_supplement.707.21
Publikováno v:
Brain research. 1217
A parasite-derived neurotrophic factor (PDNF) produced by the Chagas' disease parasite Trypanosoma cruzi binds nerve growth factor (NGF) receptor TrkA, increasing receptor autophosphorylation, and activating phosphatidylinositol 3-kinase (PI3K) and m
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::53e33e51b88d67e89ef5eeaeeeea0844
https://pubmed.ncbi.nlm.nih.gov/18502403
https://pubmed.ncbi.nlm.nih.gov/18502403