Zobrazeno 1 - 4
of 4
pro vyhledávání: '"KIDNEY TUBULES COLLECTING/pathology"'
Autor:
Raoul D. Nelson, Edith Hummler, Helle Hasager Damkier, Lance Edward Miller, Birgitte Mønster Christensen, Søren Brandt Poulsen, Jeppe Praetorius
Publikováno v:
American Journal of Physiology. Renal Physiology, vol. 310, no. 4, pp. F300-F310
Poulsen, S B, Praetorius, J, Damkier, H H, Miller, L, Nelson, R D, Hummler, E & Christensen, B M 2016, ' Reducing αENaC expression in kidney connecting tubule induces pseudohypoaldosteronism type 1 symptoms during K+ loading ', American Journal of Physiology: Renal Physiology, vol. 310, no. 4, pp. F300-F310 . https://doi.org/10.1152/ajprenal.00258.2015
Poulsen, S B, Praetorius, J, Damkier, H H, Miller, L, Nelson, R D, Hummler, E & Christensen, B M 2016, ' Reducing αENaC expression in kidney connecting tubule induces pseudohypoaldosteronism type 1 symptoms during K+ loading ', American Journal of Physiology: Renal Physiology, vol. 310, no. 4, pp. F300-F310 . https://doi.org/10.1152/ajprenal.00258.2015
Genetic inactivation of the epithelial Na+ channel α-subunit (αENaC) in the renal collecting duct (CD) does not interfere with Na+ and K+ homeostasis in mice. However, inactivation in the CD and a part of the connecting tubule (CNT) induces autosom
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::872b578644a2ebae89ef677482a31b51
https://serval.unil.ch/resource/serval:BIB_4DB30CE2E35B.P001/REF.pdf
https://serval.unil.ch/resource/serval:BIB_4DB30CE2E35B.P001/REF.pdf
Autor:
Annie Mercier Zuber, Edith Hummler, Peter M.T. Deen, Birgitte Mønster Christensen, Jean-Christophe Stehle, Johannes Loffing, Bernard C. Rossier
Publikováno v:
Christensen, B M, Mercier Zuber, A, Loffing, J, Stehle, J-C, Deen, P M T, Rossier, B C & Hummler, E 2010, ' {alpha}ENaC-Mediated Lithium Absorption Promotes Nephrogenic Diabetes Insipidus ', Journal of the American Society of Nephrology . https://doi.org/10.1681/ASN.2010070734
Journal of the American Society of Nephrology : JASN
Journal of the American Society of Nephrology, vol. 22, no. 2, pp. 253-261
Journal of the American Society of Nephrology, 22, 253-61
Journal of the American Society of Nephrology, 22, 2, pp. 253-61
Journal of the American Society of Nephrology : JASN
Journal of the American Society of Nephrology, vol. 22, no. 2, pp. 253-261
Journal of the American Society of Nephrology, 22, 253-61
Journal of the American Society of Nephrology, 22, 2, pp. 253-61
Item does not contain fulltext Lithium-induced nephrogenic diabetes insipidus (NDI) is accompanied by polyuria, downregulation of aquaporin 2 (AQP2), and cellular remodeling of the collecting duct (CD). The amiloride-sensitive epithelial sodium chann
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::61fc6541ccc3d7e15e4ed89090cc80b4
https://doi.org/10.1681/ASN.2010070734
https://doi.org/10.1681/ASN.2010070734
O gene PKHD1, mutado na doença renal policística autossômica recessiva, apresenta um padrão de splicing complexo associado a múltiplos transcritos alternativos. Neste trabalho estudamos o perfil de expressão de seu produto, poliductina. Anális
Publikováno v:
Biblioteca Digital de Teses e Dissertações da USPUniversidade de São PauloUSP.
O gene PKHD1, mutado na doença renal policística autossômica recessiva, apresenta um padrão de splicing complexo associado a múltiplos transcritos alternativos. Neste trabalho estudamos o perfil de expressão de seu produto, poliductina. Anális
