Zobrazeno 1 - 10
of 204
pro vyhledávání: '"K. de Meer"'
Autor:
Reinoud J. B. J. Gemke, Marije B. Hoos, M van der Kuip, P Ph Forget, K. de Meer, Klaas R. Westerterp
Publikováno v:
Acta Paediatrica. 92:921-927
Energy expenditure in infants with congenital heart disease, including a meta-analysis. van der Kuip M, Hoos MB, Forget PP, Westerterp KR, Gemke RJ, de Meer K. Department of Paediatrics, VU University Medical Centre, Amsterdam, The Netherlands. AIM:
Publikováno v:
van der Kuip, M, de Meer, K, Westerterp, K R & Gemke, R J 2007, ' Physical activity as a determinant of total energy expenditure in critically ill children ', Clinical Nutrition, vol. 26, no. 6, pp. 744-51 . https://doi.org/10.1016/j.clnu.2007.08.005
Clinical Nutrition, 26(6), 744-51. Churchill Livingstone
Clinical Nutrition, 26(6), 744-751. Churchill Livingstone
Clinical Nutrition, 26(6), 744-51. Churchill Livingstone
Clinical Nutrition, 26(6), 744-751. Churchill Livingstone
BACKGROUND & AIMS: For adequate nutritional support of critically ill children, knowledge of the patient's energy expenditure is required. Steady state measurement by a metabolic monitor are defined as resting energy expenditure and may underestimate
Autor:
J.t Van Den Akker, K de Meer, Jack R. Dainty, Paul Finglas, Yvo M. Smulders, Desirée E.C. Smith, R. M. Kok, Cornelis Jakobs
Publikováno v:
Analytical Biochemistry. 326:129-138
We describe a liquid chromatography (LC) tandem mass spectrometry (MS-MS) method for the determination of 5-methyltetrahydrofolic acid (5-methylTHF) and folic acid concentrations and enrichments in human plasma. It was used to study absorption and in
Publikováno v:
Journal of chromatography. B, Analytical technologies in the biomedical and life sciences, 791(1-2), 399-405. Elsevier
We describe a GC-MS and GC-c-IRMS method for the determination of labeled urea tracer enrichments in plasma as a result of combined 13C- and 15N(2)-urea infusion experiments in piglets. Urea was converted into 2-methoxypyrimidine, a stable derivative
Autor:
R.J. Heine, Annemarie Becker, Yvo M. Smulders, K de Meer, Eduard A. Struys, Lex M. Bouter, Tom Teerlink, Cornelis Jakobs, Giel Nijpels, Pieter J. Kostense, Coen D.A. Stehouwer, Jacqueline M. Dekker
Publikováno v:
European Journal of Clinical Investigation. 33:17-25
Background It is unclear whether homocysteine itself is causal in the pathogenesis of cardiovascular disease. Alternatively or additionally, the association between homocysteine and cardiovascular disease may be because of its metabolic precursor, S-
Publikováno v:
Journal of Chromatography B: Biomedical Sciences and Applications. 738:99-105
We describe a reliable method for the simultaneous determination of isotopic enrichments of [1-13C]homocysteine, [1-13C]methionine and [2H3-methyl-1-13C]methionine in human plasma. Accurate [1-13C]homocysteine calibration standards were prepared by c
Autor:
V. A. M. Gulmans, Paul J. M. Helders, J.A.J. Faber, Rudolphus Berger, K. de Meer, H. J. L. Brackel
Publikováno v:
Pediatric Pulmonology. 28:39-46
Exercise training is currently advocated as part of the treatment of patients with cystic fibrosis (CF). However, data are few that document physiologic benefits or changes in patients' perceptions of long-term training programs in children with CF.
Publikováno v:
Journal of Chromatography B: Biomedical Sciences and Applications. 729:211-216
We report a validated method for the determination of extra- and intracellular [1-(13)C]-alpha-ketoisovalerate ([1-(13)C]-KIV) enrichments by gas chromatography-mass spectrometry. Standardization curves were prepared by enzymatic oxidation of [1-(13)
Publikováno v:
Pediatric Pulmonology. 22:85-89
There is a need to judge general exercise tolerance in children with cystic fibrosis (CF) under normal daily activity conditions and -when more extensive testing is required-in an exercise laboratory in a specialized center. We investigated the repro
Publikováno v:
Thorax. 50:980-983
BACKGROUND--Exercise intolerance in patients with cystic fibrosis is commonly attributed to reduced pulmonary and nutritional status. The possible role of diminished efficiency of mitochondrial oxidative phosphorylation in relation to skeletal muscle