Zobrazeno 1 - 10
of 10
pro vyhledávání: '"K. V. Sakharova"'
Autor:
L. D. Vorobyova, T. V. Dubinina, A. O. Sablina, A. B. Demina, E. V. Ilinykh, K. V. Sakharova, I. A. Andrianova, T. V. Korotaeva, L. V. Ivanova
Publikováno v:
Современная ревматология, Vol 18, Iss 4, Pp 43-50 (2024)
Objective: validation of the Russian-language version of Ankylosing Spondylitis Quality of Life (ASQoL) specific questionnaire for patients with ankylosing spondylitis (AS). Materials and methods. Тhe study included 100 patients with a definite diag
Externí odkaz:
https://doaj.org/article/e7c9cceecce24063a1d414e420894f45
Autor:
Sh. F. Erdes, K. V. Sakharova
Publikováno v:
Современная ревматология, Vol 17, Iss 5, Pp 61-66 (2023)
The link between the HLA-B27 gene and ankylosing spondylitis (AS) has been known for almost 50 years. During this time, it has been discovered that some of the clinical manifestations of the disease are uniformly associated with the disease in almost
Externí odkaz:
https://doaj.org/article/2f5a1c77185842b58e1d42ea12ea2d4e
Publikováno v:
Современная ревматология, Vol 17, Iss 3, Pp 30-36 (2023)
The “Treat-to-target” (T2T) strategy for axial spondyloarthritis (axSpA) has made it possible to optimize therapy in most patients, but in some of them the treatment goals are not achieved.Objective: to analyze the clinical features of patients w
Externí odkaz:
https://doaj.org/article/bba8a931156345ea95db8632ffada904
Publikováno v:
Современная ревматология, Vol 15, Iss 6, Pp 67-71 (2021)
Spine involvement in ankylosing spondylitis (AS) resulting in limited mobility requires a search for accurate, quantitatively methods of assessment of the decline of its function and monitoring of its dynamics. One of the promising methods for assess
Externí odkaz:
https://doaj.org/article/a30ee2fc5157473b9eee2a00ca8be8b3
Publikováno v:
Современная ревматология, Vol 15, Iss 6, Pp 72-75 (2021)
Serum amyloid A protein A (SAA) is a normal serum protein (serving as a precursor of fibrillar tissue protein AA), synthesized in the liver and a rapidly responding marker of the acute phase of inflammation. A constant high concentration of SAA is on
Externí odkaz:
https://doaj.org/article/ea664b548d2a4c0dbcdcb7c71e716ef4
Autor:
M. V. Kireeva, E. M. Agafonova, A. E. Dimitreva, K. V. Sakharova, S. O. Krasnenko, Sh. F. Erdes
Publikováno v:
Научно-практическая ревматология, Vol 59, Iss 5, Pp 631-637 (2021)
Alopecia areata (АA) is an autoimmune multifactorial disease characterized by increased hair loss as a result of morphological and functional changes in hair follicles. АA is divided into four main forms, among which the most severe is the universa
Externí odkaz:
https://doaj.org/article/e0bb44e2ffe4405ca4f00f9107101786
Autor:
M. Yu. Krylov, I. A. Guseva, K. V. Sakharova, E. Yu. Samarkina, S. F. Erdes, N. V. Konovalova, D. A. Varlamov
Publikováno v:
Научно-практическая ревматология, Vol 60, Iss 1 (2022)
Objective. To study the involvement of the rs1800629 G/A polymorphisms of the TNF-α gene, rs1205C/T of the CRP gene, and rs12218T/C of the SAA1 gene in the predisposition to ankylosing spondylitis (AS) and their role in the formation of clinical phe
Externí odkaz:
https://doaj.org/article/9c3a742ec37344f38d81a0b9bd6b6d88
Publikováno v:
Современная ревматология, Vol 14, Iss 4, Pp 157-160 (2020)
If standard therapy with nonsteroidal anti-inflammatory drugs is ineffective, biological drugs are usually prescribed for patients with active ankylosing spondylitis. These medicines are purchased in Russia not under their trade names, but under thei
Externí odkaz:
https://doaj.org/article/171a39c171394f058b2e4c0a02af0da0
Publikováno v:
Научно-практическая ревматология, Vol 59, Iss 5, Pp 631-637 (2021)
Alopecia areata (АA) is an autoimmune multifactorial disease characterized by increased hair loss as a result of morphological and functional changes in hair follicles. АA is divided into four main forms, among which the most severe is the universa
Publikováno v:
Annals of the Rheumatic Diseases. 80:1040.1-1040
Background:Ankylosing spondylitis is a chronic systemic inflammatory disease. Inflammation and high levels of serum amyloid A (SAA) protein are predisposing factors for secondary AA amyloidosis. The role of SAA1 gene polymorphisms in AS is not well u