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pro vyhledávání: '"K. Swisshelm"'
Akademický článek
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Autor:
K. Swisshelm, R.T. Hagelstrom
A fragile site is the term given to areas of chromosomes that show a liability to DNA replicative stress resulting in gaps or breaks. Initially described in a common form of inherited intellectual disability, fragile X syndrome, over a hundred fragil
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::669846ecd0e0ba40e450f690b801239e
https://doi.org/10.1016/b978-0-12-374984-0.00553-2
https://doi.org/10.1016/b978-0-12-374984-0.00553-2
Autor:
K. Swisshelm
Anchorage-independent growth (AIG) is an acquired characteristic of cells to thrive in the absence of a natural or synthetic substrate. Multiple molecular and genetic factors contribute to AIG, including viral transformation, loss of tumor suppressor
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::39d5bf4127975ce495c488020b25e5cc
https://doi.org/10.1016/b978-0-12-374984-0.00062-0
https://doi.org/10.1016/b978-0-12-374984-0.00062-0
Akademický článek
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Publikováno v:
Cellular and molecular biology (Noisy-le-Grand, France). 49(1)
Constitutive activation of Ras or Ras-mediated signaling pathways is one of the initial steps during tumorigenesis that promotes neoplastic transformation. Recently it was reported that in Ha-Ras overexpressing MDCK cells the tight junction proteins
Publikováno v:
Endocrinology. 141(1)
Skeletal myogenic cells respond to the insulin-like growth factors (IGF-I and IGF-II) by differentiating or proliferating, which are mutually exclusive pathways. What determines which of these responses to IGF skeletal myoblast undergo is unclear. IG
Autor:
K, Swisshelm
Publikováno v:
Progress in molecular and subcellular biology. 24
Publikováno v:
Cell growthdifferentiation : the molecular biology journal of the American Association for Cancer Research. 8(6)
Retinoids mediate the normal growth of a variety of epithelial cells and may play an important role in the chemoprevention of certain malignancies. Loss of retinoic acid (RA) receptor-beta function may be an important event in mammary carcinogenesis,