Zobrazeno 1 - 9
of 9
pro vyhledávání: '"K. David Farris"'
Autor:
Monir Shababi, Caley E. Smith, Sara M. Ricardez Hernandez, Jose Marquez, Zayd Al Rawi, Eric Villalón, K. David Farris, Mona O. Garro-Kacher, Christian L. Lorson
Publikováno v:
Molecular Therapy: Methods & Clinical Development, Vol 23, Iss , Pp 23-32 (2021)
Spinal muscular atrophy with respiratory distress type 1 (SMARD1) is an autosomal recessive disorder that develops in infancy and arises from mutation of the immunoglobulin helicase μ-binding protein 2 (IGHMBP2) gene. Whereas IGHMBP2 is ubiquitously
Externí odkaz:
https://doaj.org/article/ff8c855825c74815a870d89224bf781b
Autor:
Caley E. Smith, Sara M. Ricardez Hernandez, Christian L. Lorson, Zayd Al Rawi, K. David Farris, Mona O. Garro-Kacher, Monir Shababi, Jose Marquez, Eric Villalón
Publikováno v:
Molecular Therapy - Methods & Clinical Development. 23:23-32
Spinal muscular atrophy with respiratory distress type 1 (SMARD1) is an autosomal recessive disorder that develops in infancy and arises from mutation of the immunoglobulin helicase μ-binding protein 2 (IGHMBP2) gene. Whereas IGHMBP2 is ubiquitously
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7e43815e31102b2793f4470a4c476bde
https://doi.org/10.1016/j.omtm.2021.07.008
https://doi.org/10.1016/j.omtm.2021.07.008
Autor:
Monir Shababi, Caley E. Smith, Sara M. Ricardez Hernandez, Jose Marquez, Zayd Al Rawi, Eric Villalón, K. David Farris, Mona O. Garro-Kacher, Christian L. Lorson
Publikováno v:
Molecular Therapy. Methods & Clinical Development
Molecular Therapy: Methods & Clinical Development, Vol 23, Iss, Pp 23-32 (2021)
Molecular Therapy: Methods & Clinical Development, Vol 23, Iss, Pp 23-32 (2021)
Spinal muscular atrophy with respiratory distress type 1 (SMARD1) is an autosomal recessive disorder that develops in infancy and arises from mutation of the immunoglobulin helicase μ-binding protein 2 (IGHMBP2) gene. Whereas IGHMBP2 is ubiquitously
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid_dedup__::b8d55680da5ab8faf3fcd74740ec8cf1
http://europepmc.org/articles/PMC8426477
http://europepmc.org/articles/PMC8426477
Autor:
David J. Pintel, K. David Farris
Publikováno v:
Journal of Virology. 84:1193-1197
Alternative splicing of adeno-associated virus type 2 (AAV2) P19-generated pre-mRNAs generates the small Rep proteins Rep52 and Rep40, which differ in their carboxyl termini. Both proteins are required for optimal packaging of AAV2 genomes. AAV5 Rep-
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e7ed3ba7c684ca84da44eccd79c8516e
https://doi.org/10.1128/jvi.01961-09
https://doi.org/10.1128/jvi.01961-09
Adeno-associated virus (AAV) type 2 and 5 proteins Rep52 and Rep40 were polyubiquitinated during AAV-adenovirus type 5 (Ad5) coinfection and during transient transfection in either the presence or absence of Ad5 E4orf6 and E1b-55k. Polyubiquitination
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::98afd2b4be20cc2afc141d72f27c00c3
https://europepmc.org/articles/PMC2798352/
https://europepmc.org/articles/PMC2798352/
Autor:
K. David Farris, David J. Pintel
Adeno-associated viral (AAV) capsid proteins, thought to be a rate-limiting step in the production of recombinant AAV (rAAV), are translated from spliced mRNAs. Improvement of the native AAV nonconsensus donor sequence increases splicing yet leaves t
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::642501e8df75ca20f50be7039dc75e2b
https://europepmc.org/articles/PMC2940631/
https://europepmc.org/articles/PMC2940631/
Publikováno v:
Journal of virology. 82(7)
Degradation of de novo-generated adeno-associated virus type 5 (AAV5) Rep52 and capsid proteins is part of the limited target specificity displayed by adenovirus type 5 E4Orf6-E1B-55k as part of a cullin 5-containing E3 ligase complex. Both Rep and c
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::520e455ee7b1f196ae67d15bb414e712
https://pubmed.ncbi.nlm.nih.gov/18216112
https://pubmed.ncbi.nlm.nih.gov/18216112
Autor:
K. David Farris, David J. Pintel
Publikováno v:
Human Gene Therapy. :081015093227032
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::4fa026637b8932c70dcd9984748c4dea
https://doi.org/10.1089/hgt.2008.118
https://doi.org/10.1089/hgt.2008.118
Autor:
K. David Farris, David J. Pintel
Publikováno v:
Human Gene Therapy; Dec2008, Vol. 19 Issue 12, p1421-1427, 7p