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Autor:
M P, Lambert, K L, Viola, B A, Chromy, L, Chang, T E, Morgan, J, Yu, D L, Venton, G A, Krafft, C E, Finch, W L, Klein
Publikováno v:
Journal of neurochemistry. 79(3)
In recent studies of transgenic models of Alzheimer's disease (AD), it has been reported that antibodies to aged beta amyloid peptide 1-42 (Abeta(1-42)) solutions (mixtures of Abeta monomers, oligomers and amyloid fibrils) cause conspicuous reduction
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::9880cc20830fbfed15fb58f92f4f15c3
https://pubmed.ncbi.nlm.nih.gov/11701763
https://pubmed.ncbi.nlm.nih.gov/11701763
