Zobrazeno 1 - 4
of 4
pro vyhledávání: '"K E Lines"'
Autor:
K E Lines, M Stevenson, R Mihai, I V Grigorieva, O A Shariq, K U Gaynor, J Jeyabalan, M Javid, R V Thakker
Publikováno v:
Endocrine Oncology, Vol 1, Iss 1, Pp 23-32 (2023)
Hypoxia, a primary stimulus for angiogenesis, is important for tumour proliferation and survival. The effects of hypoxia on parathyroid tumour cells, which may also be important for parathyroid autotransplantation in patients, are, however, unknown.
Externí odkaz:
https://doaj.org/article/761cd33860c04fa7b2afbaa8733011ab
Publikováno v:
Endocrine Connections, Vol 6, Iss 4, Pp 232-242 (2017)
Multiple endocrine neoplasia type 1 (MEN1) is an autosomal dominant disorder characterised by occurrence of parathyroid tumours and neuroendocrine tumours (NETs) of the pancreatic islets and anterior pituitary. The MEN1 gene, encoding menin, is a tum
Externí odkaz:
https://doaj.org/article/f4f4891489d34467b28c78b9be8c5bb5
Autor:
G V, Walls, M, Stevenson, K E, Lines, P J, Newey, A A C, Reed, M R, Bowl, J, Jeyabalan, B, Harding, K J, Bradley, S, Manek, J, Chen, P, Wang, B O, Williams, B T, Teh, R V, Thakker
Publikováno v:
Oncogene
The hyperparathyroidism-jaw tumour (HPT-JT) syndrome is an autosomal dominant disorder characterized by occurrence of parathyroid tumours, often atypical adenomas and carcinomas, ossifying jaw fibromas, renal tumours and uterine benign and malignant
Publikováno v:
Molecular and Cellular Endocrinology
Pituitary neoplasias can occur as part of a complex inherited disorder, or more commonly as sporadic (non-familial) disease. Studies of the molecular and genetic mechanisms causing such pituitary tumours have identified dysregulation of >35 genes, wi