Zobrazeno 1 - 8
of 8
pro vyhledávání: '"K A Gravel"'
Publikováno v:
The Journal of Immunology. 156:2143-2154
CBA/N mice carry an X-linked immunodeficiency (xid) due to a point mutation in the Bruton's tyrosine kinase (btk) gene. xid mice have a smaller peripheral B cell pool than normal animals, lack CD5+ B cells (B1), and are hyporesponsive to mitogenic an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::33238afe9a0e30f9e98bc41995a108cb
https://doi.org/10.4049/jimmunol.156.6.2143
https://doi.org/10.4049/jimmunol.156.6.2143
Publikováno v:
The Journal of Immunology. 155:3453-3463
Normal B cells responsive to thymus independent-type 1 Ags (TI-1) are resistant to low doses of ionizing radiation in vivo (200-300 cGy), compared with TI-1 responsive B cells of mice with the CBA/N X-linked immunodeficiency (xid). This difference in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::f0839e93bd57e76681d9017c0922e901
https://doi.org/10.4049/jimmunol.155.7.3453
https://doi.org/10.4049/jimmunol.155.7.3453
Publikováno v:
The Journal of Immunology. 155:2533-2544
Small resting B cells do not support a productive vesicular stomatitis virus (VSV) infection, but are induced by B cell activators to become fully permissive for VSV replication. Nonpermissive B cell populations restrict VSV expression at multiple po
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0db162d352ec9ef95aaf27cd885a1a7c
https://doi.org/10.4049/jimmunol.155.5.2533
https://doi.org/10.4049/jimmunol.155.5.2533
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 156(6)
CBA/N mice carry an X-linked immunodeficiency (xid) due to a point mutation in the Bruton's tyrosine kinase (btk) gene. xid mice have a smaller peripheral B cell pool than normal animals, lack CD5+ B cells (B1), and are hyporesponsive to mitogenic an
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::c0a2fb1b89c20ec6c2142551099700b3
https://pubmed.ncbi.nlm.nih.gov/8690903
https://pubmed.ncbi.nlm.nih.gov/8690903
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 155(7)
Normal B cells responsive to thymus independent-type 1 Ags (TI-1) are resistant to low doses of ionizing radiation in vivo (200-300 cGy), compared with TI-1 responsive B cells of mice with the CBA/N X-linked immunodeficiency (xid). This difference in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::12cb7c535d8f4fbcce7fd8d775c339dd
https://pubmed.ncbi.nlm.nih.gov/7561040
https://pubmed.ncbi.nlm.nih.gov/7561040
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 155(5)
Small resting B cells do not support a productive vesicular stomatitis virus (VSV) infection, but are induced by B cell activators to become fully permissive for VSV replication. Nonpermissive B cell populations restrict VSV expression at multiple po
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::30cd35b936d57a333031798ad5afe3ce
https://pubmed.ncbi.nlm.nih.gov/7650383
https://pubmed.ncbi.nlm.nih.gov/7650383
Publikováno v:
The Journal of Immunology. 141:4243-4249
Mouse B cells are stimulated to proliferate by Fab'2 fragments of rabbit anti-mouse Ig antibodies. Proliferation is inhibited, however, in the presence of IgG anti-mouse Ig. We have previously shown that this inhibition is mediated by binding of the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::bb7e32441d68d816ac52ccda6948e4d6
https://doi.org/10.4049/jimmunol.141.12.4243
https://doi.org/10.4049/jimmunol.141.12.4243
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 141(12)
Mouse B cells are stimulated to proliferate by Fab'2 fragments of rabbit anti-mouse Ig antibodies. Proliferation is inhibited, however, in the presence of IgG anti-mouse Ig. We have previously shown that this inhibition is mediated by binding of the
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::95d0359a88683b440cb14a318d1aca85
https://pubmed.ncbi.nlm.nih.gov/2974056
https://pubmed.ncbi.nlm.nih.gov/2974056