Zobrazeno 1 - 9
of 9
pro vyhledávání: '"K A, Maslonek"'
Publikováno v:
The Journal of Immunology. 152:5845-5852
The present study examined the role of macrophage-derived nitric oxide in the suppressive effect of in vivo morphine administration on Con A-stimulated proliferation of splenic lymphocytes in rats. The results showed that concentrations of nitrite ar
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 277(2)
The present study examined the involvement of the sympathetic nervous system and the hypothalamic-pituitary-adrenal axis in the immunomodulatory effects of acute morphine treatment in rats. Chlorisondamine, a ganglionic blocker, was used to assess th
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 276(2)
The present study assessed the involvement of opioid receptors both in the central nervous system (CNS) and in the periphery (i.e., on immunocytes) in the immune alterations produced by acute morphine treatment in rats. The first experiment showed th
Publikováno v:
The Journal of pharmacology and experimental therapeutics. 272(2)
Previous research by our laboratory demonstrated that in vivo administration of morphine to rats suppresses concanavalin-A (Con-A)-stimulated proliferation of splenic lymphocytes in a dose-dependent, naltrexone-reversible manner. More recently, we sh
Publikováno v:
Advances in experimental medicine and biology. 373
Publikováno v:
Advances in experimental medicine and biology. 373
Publikováno v:
Advances in experimental medicine and biology. 373
Publikováno v:
Journal of immunology (Baltimore, Md. : 1950). 152(12)
The present study examined the role of macrophage-derived nitric oxide in the suppressive effect of in vivo morphine administration on Con A-stimulated proliferation of splenic lymphocytes in rats. The results showed that concentrations of nitrite ar
Publikováno v:
Behavioral neuroscience. 105(3)
The effect of propranolol, a beta-adrenergic receptor antagonist, on the suppression of splenic mitogenic responsiveness induced by an aversive conditioned stimulus (CS) was evaluated in rats. Experiment 1 demonstrated that propranolol administration