Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Justyna Brańska-Januszewska"'
Autor:
Klaudia Kijowska-Sieroń, Magdalena Wojtanowska-Kaczka, Magdalena Babuśka-Roczniak, Wojciech Roczniak, Elżbieta Cipora, Justyna Brańska-Januszewska, Anna Roczniak-Zubrycka, Magdalena Konieczny
Publikováno v:
Pomeranian Journal of Life Sciences. 66:5-10
Introduction: Speed of movement refers to the max. speed of moves that an individual is able to perform, associated with the optimal activation of muscle fibers. It increases until the age of 15 in girls and 18 in boys, with the fastest rate 6–14 y
Autor:
Joanna Sutkowska-Skolimowska, Justyna Brańska-Januszewska, Jakub W. Strawa, Halina Ostrowska, Malwina Botor, Katarzyna Gawron, Anna Galicka
Publikováno v:
International Journal of Molecular Sciences; Volume 23; Issue 18; Pages: 10341
Osteogenesis imperfecta (OI) is a heterogeneous connective tissue disease mainly caused by structural mutations in type I collagen. Mutant collagen accumulates intracellularly, causing cellular stress that has recently been shown to be phenotype-rela
Autor:
Ewa Chabielska, Marian Tomasiak, Agata Golaszewska, Justyna Brańska-Januszewska, Natalia Marcinczyk, Tomasz Misztal, Tomasz Rusak
Publikováno v:
Frontiers in Physiology, Vol 11 (2020)
Frontiers in Physiology
Frontiers in Physiology
Background: Recent studies indicate that aquaporin (AQP) water channels have a regulatory function in human platelet secretion and in procoagulant response of murine platelets. However, the engagement of AQPs in morphological changes, procoagulant re
Autor:
Magdalena Konieczny, Magdalena Babuśka-Roczniak, Magdalena Wojtanowska, Wojciech Roczniak, Elżbieta Cipora, Justyna Brańska Januszewska, Anna Roczniak Zubrzycka, Joanna Oświęcimska
Publikováno v:
Medycyna Ogólna i Nauki o Zdrowiu. 24:70-75
Autor:
Adrian Chabowski, Agnieszka Mikłosz, Małgorzata Żendzian-Piotrowska, Bartłomiej Łukaszuk, Justyna Brańska-Januszewska, Halina Ostrowska
Publikováno v:
Journal of Cellular Physiology. 232:2373-2386
The Akt substrate of 160 kDa (AS160) is a key regulator of GLUT4 translocation from intracellular depots to the plasma membrane in myocytes. Likely, AS160 also controls LCFAs transport, which requires relocation of fatty acid transporters. The aim of
Autor:
Tomasz Rusak, Marta Bruczko, Beata Szynaka, Marta Gąsowska, Agata Golaszewska, Justyna Brańska-Januszewska, Tomasz Misztal, Marian Tomasiak
Publikováno v:
Acta biochimica Polonica. 65(4)
This study was undertaken to establish the presence and the role of aquaporins (AQPs) in human platelets. Immunodetection with polyclonal antibodies and fluorescent microscopy suggest the presence of AQP isoforms – 0–7 and 9–12 – localized (i
Publikováno v:
Blood coagulationfibrinolysis : an international journal in haemostasis and thrombosis. 28(6)
Rise in mean platelet volume (MPV) has been demonstrated to be associated with increased platelet reactivity. In diabetes patients, augmented MPV was proposed to contribute to increased risk of thrombotic complications. Therefore, the aim of this stu
Publikováno v:
Thrombosis Research. 134:192-198
Using patients with polycythemia vera (PV) as an experimental model, we evaluated the impact of clot retraction (CR) and architecture of the clot on fibrinolysis. We studied the kinetics of clot retraction and the fibrinolysis rate in whole blood fro
Autor:
Tomasz Misztal, Tomasz Rusak, M. M. Tomasiak-Lozowska, Justyna Brańska-Januszewska, Anna Bodzenta-Lukaszyk, Marian Tomasiak
Publikováno v:
Allergy. 72(2)
The aim of this study was to assess whether steroid-naive asthma modulates hemostasis. We evaluated the clot retraction rate (CRR), fibrinolysis rate (FR), clot density (CD) (by confocal microscopy), plasma levels of plasminogen activator inhibitor (
Publikováno v:
Free radical biologymedicine. 89
We tested the hypothesis that in vitro peroxynitrite (ONOO(-), a product of activated inflammatory cells) may affect fibrinolysis in human blood through the reduction of platelet-related fibrinolysis resistance. It was found that ONOO(-) (25-300 µM)