Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Junsa Geng"'
Autor:
Junsa Geng, Yan Mei, Guangli Suo, Zhongjuan Xu, Shumang Zhang, Yu Liang, Yanzhen Yu, Tongqian Xiao, Yong Qiao, Bin Wang, Jiali Wei, Qun Dong
Publikováno v:
Journal of Tissue Engineering and Regenerative Medicine. 13:1759-1769
Amyloid plaque is a typical feature of Alzheimer's disease (AD) and is one of the targets for AD therapy. Membrane metalloendopeptidase (MME) and insulin-degrading enzyme (IDE) are two types of proteases that could cleave beta-amyloid (Aβ) peptides
Autor:
Guangli Suo, Yanzhen Yu, Tongqian Xiao, Zhongjuan Xu, Junsa Geng, Qun Dong, Yong Qiao, Hai Zhang, Yu Liang
Publikováno v:
BMB Reports
Epithelial-mesenchymal transition (EMT) is widely-considered to be a modulating factor of anoikis and cancer metastasis. We found that, in MDA-MB-231 cells, TP53I11 (tumor protein P53 inducible protein 11) suppressed EMT and migration in vitro, and i
Autor:
Guangli Suo, Yuanshuai Zhou, Tongqian Xiao, Junsa Geng, Hong Qiao, Zhongjuan Xu, Yu Liang, Hai Zhang
Publikováno v:
IUBMB Life. 71:183-191
Extracellular matrix (ECM)-independent survival is an essential prerequisite for tumor metastasis, and a hallmark of epithelial cancer stem cells and epithelial-mesenchymal transition (EMT). Here, we found that loss of TP53I11 enhanced, and overexpre
Autor:
Yan Mei, Guangli Suo, Yanzhen Yu, Tongqian Xiao, Junsa Geng, Hong Qiao, Yong Qiao, Shulan Hou, Jianwu Dai, Yu Liang, Qun Dong, Bin Wang, Zhongjuan Xu
Publikováno v:
Biomaterials. 227:119573
Umbilical cord mesenchymal stem cells (UCMSCs) have shown great potentials in regenerative medicine for their extensive sources, multilineage differentiation potential, low immunogenicity and self-renewal ability. However, the clinical application of
Autor:
Tongqian, Xiao, Zhongjuan, Xu, Yuanshuai, Zhou, Hai, Zhang, Junsa, Geng, Yu, Liang, Hong, Qiao, Guangli, Suo
Publikováno v:
IUBMB life. 71(2)
Extracellular matrix (ECM)-independent survival is an essential prerequisite for tumor metastasis, and a hallmark of epithelial cancer stem cells and epithelial-mesenchymal transition (EMT). Here, we found that loss of TP53I11 enhanced, and overexpre
Autor:
Yu-Guang Zhou, Huajie Zhang, Guangli Suo, Yu-lai Liang, Junsa Geng, Hong Qiao, Ziying Xu, Tengfei Xiao
Extracellular matrix (ECM)-independent survival is an essential prerequisite for tumor metastasis and a hallmark of epithelial cancer stem cells and epithelial-mesenchymal transition (EMT). We found that, in MCF10A and MDA-MB-231 cells, loss of TP53I
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::cf9b9a5385d73733e6e526b0bf35e122
https://doi.org/10.1101/301499
https://doi.org/10.1101/301499