Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Junqin, Sheng"'
Publikováno v:
Cellular Physiology and Biochemistry, Vol 48, Iss 4, Pp 1675-1693 (2018)
Background/Aims: Disrupted mitochondrial dynamics, including excessive mitochondrial fission and mitophagy arrest, has been identified as a pathogenic factor in diabetic nephropathy (DN), although the upstream regulatory signal for mitochondrial fiss
Externí odkaz:
https://doaj.org/article/55ef232159cf45698ef1e12c2e247f7b
Publikováno v:
Cellular Physiology and Biochemistry, Vol 46, Iss 4, Pp 1508-1524 (2018)
Background/Aims: Amniotic fluid stem cells (AFSCs) transplantation is a promising therapeutic strategy for diabetic nephropathy. Sirtuin3 (SIRT3) is a novel mitochondrial protective factor. In the present study, we aimed to investigate whether SIRT3
Externí odkaz:
https://doaj.org/article/f93464d104334f5d9da2330d060d45f2
Publikováno v:
Biochemical and Biophysical Research Communications. 559:141-147
Diabetic nephropathy (DN) is the primary cause of end-stage renal disease, and renal tubular cell dysfunction contributes to the pathogenesis of many kidney diseases. Our previous study demonstrated that dual-specificity protein phosphatase 1 (DUSP1)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::3c3d040aafcf555a632abfe74c510492
https://doi.org/10.1016/j.bbrc.2021.04.032
https://doi.org/10.1016/j.bbrc.2021.04.032
Publikováno v:
Toxicology in Vitro. 86:105482
Diabetic nephropathy (DN) is the direct cause of end-stage renal disease, and nephritic inflammation plays a role in its growth and advancement. Aberrant expression of long non-coding RNAs (lncRNAs) correlates with many diseases, including DN. In thi
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::faad1435a52a41116fc2da543941e94d
https://doi.org/10.1016/j.tiv.2022.105482
https://doi.org/10.1016/j.tiv.2022.105482
Publikováno v:
Journal of Cellular Physiology. 234:3043-3057
Excessive mitochondrial fission has been identified as the pathogenesis of diabetic nephropathy (DN), although the upstream regulatory signal for mitochondrial fission activation in the setting of DN remains unknown. In the current study, we found th
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::e15702f78f94ea721b359fb331ea7a9c
https://doi.org/10.1002/jcp.27124
https://doi.org/10.1002/jcp.27124
Publikováno v:
Cellular Physiology and Biochemistry, Vol 48, Iss 4, Pp 1675-1693 (2018)
Background/Aims: Disrupted mitochondrial dynamics, including excessive mitochondrial fission and mitophagy arrest, has been identified as a pathogenic factor in diabetic nephropathy (DN), although the upstream regulatory signal for mitochondrial fiss
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::265e77483732c6b8a377ba12e51af467
https://www.karger.com/Article/FullText/492292
https://www.karger.com/Article/FullText/492292
Publikováno v:
Cellular Physiology and Biochemistry, Vol 46, Iss 4, Pp 1508-1524 (2018)
Background/Aims: Amniotic fluid stem cells (AFSCs) transplantation is a promising therapeutic strategy for diabetic nephropathy. Sirtuin3 (SIRT3) is a novel mitochondrial protective factor. In the present study, we aimed to investigate whether SIRT3
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::50da960277583bf36c30a6d5df8add0b
https://doi.org/10.1159/000489194
https://doi.org/10.1159/000489194
Publikováno v:
Scientific Reports
1,5-anhydroglucitol (1,5-AG), uric acid and urinary proteins are excreted into the urine with increasing glucosuria. In the present retrospective study we analyzed whether these factors could be used as indicators for type 2 diabetes mellitus (T2DM)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::c44ab285284413d925b975b44978d6cd
https://pubmed.ncbi.nlm.nih.gov/28281675
https://pubmed.ncbi.nlm.nih.gov/28281675