Zobrazeno 1 - 10
of 263
pro vyhledávání: '"Jungermann, Kurt"'
Autor:
Püschel, Gerhard, Jungermann, Kurt
Publikováno v:
Progr Liver Dis 12 (1994). 12:19-46
Content: Architecture of the liver acinus Functional zonation of the liver acinus Topological organization of metabollc regulation in the acinus Topological organization of defense and organ structure regulation in the acinus
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2011/5127/
Publikováno v:
European Journal of Biochemistry 207. (2):399-411
Content: Anatomy of hepatic innervation In vivo studies on the role of hepatic nerves Effects of hepatic nerves in isolated perfused liver Mechanism of action of sympathetic hepatic nerves
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2011/5134/
Publikováno v:
FEBS Letters 372 (1995), 1, p. 108-112, DOI 10.1016/0014-5793(95)00883-B, ISSN 0014-5793
Human anaphylatoxin C3a had previously been shown to increase glycogenolysis in perfused rat liver and prostanoid formation in rat liver macrophages. Surprisingly, human C5a, which in other systems elicited stronger responses than C3a, did not increa
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2010/4590/
Publikováno v:
Hepatoloy. - ISSN 0270-9139. - 22 (1995), 5, p. 1577 - 1583
Prostaglandins, released from Kupffer cells, have been shown to mediate the increase in hepatic glycogenolysis by various stimuli such as zymosan, endotoxin, immune complexes, and anaphylotoxin C3a involving prostaglandin (PG) receptors coupled to ph
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2008/1669/
Autor:
Neuschäfer-Rube, Frank, DeVries Christa, Hänecke, Kristina, Jungermann, Kurt, Püschel, Gerhard
Publikováno v:
FEBS Letters 351 (1994), 1, p. 119-122, DOI 10.1016/0014-5793(94)00837-X
Rat hepatocytes have previously been reported to possess prostaglandin E₂ receptors of the EP₃-type (EP₃-receptors) that inhibit glucagonstimulated glycogenolysis by decreasing cAMP. Here, the isolation of a functional EP₃ϐ receptor cDNA clo
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2010/4583/
Publikováno v:
Hepatology. - 19 (1994), 5, pp. 1198-1207. - ISSN 0270-9139
The site of confluence of the artery and the portal vein in the liver still appears to be controversial. Anatomical studies suggested a presinusoidal or an intrasinusoidal confluence in the first, second or even final third of the sinusoids. The obje
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2008/1670/
Publikováno v:
European Journal of Biochemistry 217 (1993), 1, p. 305-311, DOI 10.1111/j.1432-1033.1993.tb18247.x, ISSN 0014-2956
In perfused rat livers, infusion of prostaglandin F₂α (PGF₂α) or noradrenaline increased glucose and lactate output and reduced flow. Glucagon increased glucose output and decreased lactate output without influence on flow. Infusion of phorbol
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2010/4588/
Publikováno v:
European Journal of Biochemistry 218 (1993), 3, p. 1083-1089, DOI 10.1111/j.1432-1033.1993.tb18468.x, ISSN 0014-2956
Prostaglandin E₂ has been reported both to stimulate glycogen-phosphorylase activity (glycogenolytic effect) and to inhibit the glucagon-stimulated glycogen-phosphorylase activity (antiglycogenolytic effect) in rat hepatocytes. It was the purpose o
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2010/4585/
Publikováno v:
European Journal of Biochemistry 211 (1993), 1-2, p. 163-169, DOI 10.1111/j.1432-1033.1993.tb19883.x, ISSN 0014-2956
Prostaglandin (PG)F₂α has previously been shown to increase glucose output from perfused livers and isolated hepatocytes, where it stimulated glycogen phosphorylase via an inositol-trisphosphatedependent signal pathway. In this study, PGF₂α bin
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2010/4586/
Publikováno v:
European Journal of Biochemistry 196 (1991), 2, p. 525-530, DOI 10.1111/j.1432-1033.1991.tb15845.x, ISSN 0014-2956
Rat serum, in which the complement sytem had been activated by incubation with zymosan, increased the glucose and lactate output, and reduced and redistributed the flow in isolated perfused rat liver clearly more than the control serum. Heat inactiva
Externí odkaz:
http://opus.kobv.de/ubp/volltexte/2010/4589/