Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Jung Hyun Bu"'
Autor:
Myoung Sup Shim, Jung Hyun Bu, Won-Kyu Ju, Tae Lim Park, Robert N. Weinreb, Sangphil Ahn, Keunyoung Kim
Publikováno v:
Biochemical and biophysical research communications, vol 503, iss 4
Coenzyme Q10 (CoQ(10)) protects retinal ganglion cells (RGCs) in experimental retinal ischemia and glaucoma by scavenging reactive oxygen species. We tested whether a diet supplemented with ubiquinol, the reduced form of CoQ(10), promotes RGC surviva
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::202b549bdfd5e4dbd9fb9978a1758f84
https://escholarship.org/uc/item/9sb2h86w
https://escholarship.org/uc/item/9sb2h86w
Autor:
Hye Seung Nam, Won-Kyu Ju, Robert N. Weinreb, Mark H. Ellisman, Seung Won Jeong, Jung Hyun Bu, Myoung Sup Shim, Tae Lim Park, Keunyoung Kim
Publikováno v:
Cell Death & Disease
Cell death & disease, vol 9, iss 3
Cell Death and Disease, Vol 9, Iss 3, Pp 1-18 (2018)
Cell death & disease, vol 9, iss 3
Cell Death and Disease, Vol 9, Iss 3, Pp 1-18 (2018)
Glaucoma is characterized by a progressive loss of retinal ganglion cells and their axons, but the underlying biological basis for the accompanying neurodegeneration is not known. Accumulating evidence indicates that structural and functional abnorma
Publikováno v:
Experimental Neurobiology
The major defining pathological hallmarks of Alzheimer's disease (AD) are the accumulations of Aβ in senile plaques and hyperphosphorylated tau in neurofibrillary tangles and neuropil threads. Recent studies indicate that rather than these insoluble
Autor:
Jung Hyun Bu, David E. Kang, A-Rong Jung, Jung A. Woo, Madepalli K. Lakshmana, Anaid Bedrossian, Sun Ah Park, Yeory Lim, Inhee Mook-Jung, Edward H. Koo
Publikováno v:
Alzheimer's & Dementia. 8
Autor:
Yoon Lim, A R Jung, Jung Hyun Bu, A Bedrossian, David E. Kang, Edward H. Koo, Madepalli K. Lakshmana, Jung A. Woo, Sun Ah Park, Inhee Mook-Jung
Publikováno v:
Cell death and differentiation. 19(9)
Neurodegeneration associated with amyloid β (Aβ) peptide accumulation, synaptic loss, neuroinflammation, tauopathy, and memory impairments encompass the pathophysiological features of Alzheimer's disease (AD). We previously reported that the scaffo