Zobrazeno 1 - 10
of 19
pro vyhledávání: '"Jun Tanikawa"'
Autor:
Hiroshi Nakagawa, Hideharu Hase, Seishiro Sawamura, Masayuki X. Mori, Kyosuke Hino, Akito Nakao, Ryuji Inoue, Jun Tanikawa, Shigeki Kiyonaka, Mitsuru Hirano, Yaopeng Hu, Rachapun Rotrattanadumrong, Yasuo Mori, Motohiro Nishida, Ryu Nagata, Masahiko Hatano, Yoshinori Takada, Tetsuya Kawamura
Publikováno v:
Molecular Pharmacology. 89:348-363
Transient receptor potential canonical (TRPC) proteins form Ca(2+)-permeable cation channels activated upon stimulation of metabotropic receptors coupled to phospholipase C. Among the TRPC subfamily, TRPC3 and TRPC6 channels activated directly by dia
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::ca7503c04cc9e9d671ffebcb7e1d3ba0
https://doi.org/10.1124/mol.115.102863
https://doi.org/10.1124/mol.115.102863
Publikováno v:
Journal of Biological Chemistry. 279:44582-44589
Recently we have shown that the c-myb proto-oncogene product (c-Myb) is degraded in response to Wnt-1 signaling via the pathway involving TAK1 (transforming growth factor-beta-activated kinase), HIPK2 (homeodomain-interacting protein kinase 2), and N
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f630e848f5ba3fd8dd7a1d6ef16aef2b
https://doi.org/10.1074/jbc.m407831200
https://doi.org/10.1074/jbc.m407831200
Autor:
Hiroki Ito, Matiullah Khan, Jun Tanikawa, Emi Ichikawa-Iwata, Shunsuke Ishii, Hiroshi Akimaru, Teruaki Nomura, Chie Kanei-Ishii
Publikováno v:
Journal of Biological Chemistry. 279:16715-16726
The c-myb proto-oncogene product (c-Myb) regulates proliferation of hematopoietic cells by inducing the transcription of a group of target genes. Removal or mutations of the negative regulatory domain (NRD) in the C-terminal half of c-Myb leads to in
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::efcb1ccd0b21969b7cb76953522db51e
https://doi.org/10.1074/jbc.m313069200
https://doi.org/10.1074/jbc.m313069200
Autor:
Shunsuke Ishii, Yongsok Kim, Jun Tanikawa, Jun Ninomiya-Tsuji, Emi Ichikawa-Iwata, Kenji Kokura, Tohru Ishitani, Satoshi Kishida, Kunihiro Matsumoto, Chie Kanei-Ishii, Toshihiro Kurahashi, Teruaki Nomura
Publikováno v:
Genes & Development. 18:816-829
The c-myb proto-oncogene product (c-Myb) regulates both the proliferation and apoptosis of hematopoietic cells by inducing the transcription of a group of target genes. However, the biologically relevant molecular mechanisms that regulate c-Myb activ
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::177ef073cbb41a7537838820d45d7288
https://doi.org/10.1101/gad.1170604
https://doi.org/10.1101/gad.1170604
Publikováno v:
Blood Cells, Molecules, and Diseases. 27:479-482
Heat shock proteins (HSPs) act as chaperones and play important roles during cellular proliferation and apoptosis. Heat shock factors (HSFs) mediate transcriptional induction of HSP genes. Among multiple heat shock transcription factors (HSFs) in ver
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::936dbe7987457dd39811b449f41d8813
https://doi.org/10.1006/bcmd.2001.0408
https://doi.org/10.1006/bcmd.2001.0408
Autor:
Shunsuke Ishii, John C. Reed, Jun Tanikawa, Chie Kanei-Ishii, Akira Nakai, Emi Ichikawa-Iwata, Shu-ichi Matsuzawa
Publikováno v:
Journal of Biological Chemistry. 275:15578-15585
Expression of heat shock proteins (HSPs) is controlled by heat shock transcription factors (HSFs). Vertebrates express multiple HSFs whose activities may be regulated by distinct signals. HSF3 is specifically activated in unstressed proliferating cel
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::7ab27123c4261608244b05a5ba8c2d22
https://doi.org/10.1074/jbc.m000372200
https://doi.org/10.1074/jbc.m000372200
Autor:
Zhongwei Jiang, Keiichiro Uchi, Takashige Namima, Seiichi Orikasa, Seiji Chonan, Mami Tanaka, Kentaro Hirano, Jun Tanikawa
Publikováno v:
International Journal of Applied Electromagnetics and Mechanics. 10:45-62
This paper is concerned with the development of an artificial urethral valve for treating urinary incontinence, which is driven by shape memory alloy actuators. Two types of valves, one with hand-drum form and the other with cylindrical configuration
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::8a1d1baa8b3d269a7b02e22da6a6e236
https://doi.org/10.3233/jae-1999-128
https://doi.org/10.3233/jae-1999-128
Autor:
Jun Tanikawa, Mitsuhiko Ikura, Toshio Furuya, Masanori Osawa, Toshiyuki Tanaka, Toshiyasu Mase, Mark B. Swindells
Publikováno v:
Journal of Molecular Biology. 276:165-176
The solution structure of calcium-bound calmodulin (CaM) complexed with an antagonist, N-(6-aminohexyl)-5-chloro-1-naphthalenesulfonamide (W-7), has been determined by multidimensional NMR spectroscopy. The structure consists of one molecule of W-7 b
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0a9abd9b87e1638b0712f0e0e475549e
https://doi.org/10.1006/jmbi.1997.1524
https://doi.org/10.1006/jmbi.1997.1524
Autor:
Mami Tanaka, Seiji Chonan, Keiichiro Uchi, Jun Tanikawa, Zhongwei Jiang, Takasige Namima, Kentaro Hirano, Seiichi Orikasa
Publikováno v:
TRANSACTIONS OF THE JAPAN SOCIETY OF MECHANICAL ENGINEERS Series C. 64:1229-1236
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::08a171ce793976ea3de5dfabb0bded17
https://doi.org/10.1299/kikaic.64.1229
https://doi.org/10.1299/kikaic.64.1229
Autor:
Y. Tanahashi, Seiji Chonan, Toshiyuki Takagi, Seiichi Orikasa, Junji Tani, Jun Tanikawa, Zhongwei Jiang, Mami Tanaka
Publikováno v:
Smart Materials and Structures. 6:410-414
The development of an artificial urethral valve for the treatment of urinary incontinence which occurs frequently in the aged is described. The prototype urethral valve is assembled in hand-drum form with four thin shape memory alloy (SMA) (nickel -
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::3232bc72f45d5bc014209031b78eb6d0
https://doi.org/10.1088/0964-1726/6/4/004
https://doi.org/10.1088/0964-1726/6/4/004