Zobrazeno 1 - 9
of 9
pro vyhledávání: '"Jun Kadekawa"'
Publikováno v:
Japanese Journal of Neurosurgery. 22:216-220
Publikováno v:
The Japanese Journal of Rehabilitation Medicine. 39:451-456
運動ニューロン疾患の一病型であるKennedy-Alter-Sung症候群に対する筋力トレーニングの有効性を検討した.対象は遺伝子診断により確定した男性7名で,筋力トレーニングと基本動作訓練を指
Autor:
Misako Kaido, Nobuyuki Shirahata, Tomoya Nishimura, Hiroo Yoshikawa, Noriaki Hattori, Saburo Sakoda, Jun Kadekawa, Yasuko Ogawa, Takehiko Yanagihara, Harutoshi Fujimura
Publikováno v:
Acta Neuropathologica. 94:617-622
The recognition of mutations in the copper/zinc superoxide dismutase (SOD1) gene in familial amyotrophic lateral sclerosis (FALS) has been a landmark in ALS research. We report a clinicopathological study of a female patient with FALS showing a two b
Publikováno v:
Neurology. 49:837-841
We measured oxygen consumption in the exercising lower limb by using noninvasive tissue oximetry with the near-infrared spectra of hemoglobin in the quadriceps muscle during bicycle ergometer exercise in four normal controls and three patients with c
Publikováno v:
The Japanese Journal of Rehabilitation Medicine. 34:134-137
非侵襲ハンディ酸素モニタを用いて,mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes (MELAS)患者のエルゴ負荷時の血液量と組織酸素レベルの測定を行った.正常コントロールと
Autor:
Misa, Nakano, Yasushi, Takase, Chikao, Tatsumi, Atsuko, Miura, Tatsuo, Matsuyama, Jun, Kadekawa, Hiroo, Yoshikawa
Publikováno v:
Rinsho shinkeigaku = Clinical neurology. 42(1)
A 64 year-old man began to feel numbness on his bilateral feet in 1990. He was diagnosed as diabetes mellitus with a high fasting glucose level of 580 mg/dl in 1993 and he received oral hypoglycemic agents. Since then, his blood glucose levels had be
Publikováno v:
Journal of the neurological sciences. 162(1)
We evaluated the effect of coenzyme Q10 supplementation to two patients with mitochondrial myopathy, encephalopathy, lactic acidosis, and stroke-like episodes (MELAS) by using noninvasive tissue oximetry with near-infrared spectra of hemoglobin from
Publikováno v:
Neurology. 51:641-642
Myotonic dystrophy (MD) is a dominantly inherited disease.1 The genetic abnormality has been localized to chromosome 19q13.3, where expanded CTG repeat is found in the 3′ untranslated region encoding the putative serine-threonine protein kinase.2 M
Publikováno v:
Neurology. 51:1239.1-1239
Reply from the Authors: We thank Drs. Quaresima and Ferrari for their comments.1 The rapid advance in medical technology is such that the most current technology may …