Zobrazeno 1 - 10
of 10
pro vyhledávání: '"Jumpei Morita"'
Autor:
Masako Yokota, Yukiho Kobayashi, Jumpei Morita, Hiroyuki Suzuki, Yoshihide Hashimoto, Yoshihiro Sasaki, Kazunari Akiyoshi, Keiji Moriyama
Publikováno v:
PLoS ONE, Vol 9, Iss 7, p e101693 (2014)
Apert syndrome is an autosomal dominantly inherited disorder caused by missense mutations in fibroblast growth factor receptor 2 (FGFR2). Surgical procedures are frequently required to reduce morphological and functional defects in patients with Aper
Externí odkaz:
https://doaj.org/article/337958229cf14b40b0911c6afc15223a
Autor:
Michio Otsuki, Jumpei Morita
Publikováno v:
The European physical journal. E, Soft matter. 44(8)
We numerically investigate the stress distribution of a paste when an external oscillation is applied. The paste memorizes the oscillation through plastic deformation. Due to the plastic deformation, the residual stress remains after the oscillation,
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::31b5eb6acdb1b9f3b8326df68ed7b2da
https://pubmed.ncbi.nlm.nih.gov/34409512
https://pubmed.ncbi.nlm.nih.gov/34409512
Autor:
Tsukasa Watanabe, Kojiro Yamaji, Momotoshi Shiga, Jumpei Morita, Kentaro Ono, Kaori Gunjigake, Tatsuo Kawamoto, Mitsushiro Nakatomi, Keiji Moriyama
Publikováno v:
Developmental Dynamics. 247:1175-1185
Background Apert syndrome is characterized by craniosynostosis and bony syndactyly of the hands and feet. The cause of Apert syndrome is a single nucleotide substitution mutation (S252W or P253R) in fibroblast growth factor receptor 2 (FGFR2). Clinic
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::057fb01eeae799d7293388b19b10379d
https://doi.org/10.1002/dvdy.24673
https://doi.org/10.1002/dvdy.24673
Autor:
Kojiro, Yamaji, Jumpei, Morita, Tsukasa, Watanabe, Kaori, Gunjigake, Mitsushiro, Nakatomi, Momotoshi, Shiga, Kentaro, Ono, Keiji, Moriyama, Tatsuo, Kawamoto
Publikováno v:
Developmental dynamics : an official publication of the American Association of Anatomists. 247(11)
Apert syndrome is characterized by craniosynostosis and bony syndactyly of the hands and feet. The cause of Apert syndrome is a single nucleotide substitution mutation (S252W or P253R) in fibroblast growth factor receptor 2 (FGFR2). Clinical experien
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=pmid________::cfbdc1a74b8a2c5ea4e689d58e3d40fd
https://pubmed.ncbi.nlm.nih.gov/30251381
https://pubmed.ncbi.nlm.nih.gov/30251381
Publikováno v:
European journal of orthodontics. 39(3)
Background and objectives Relaxin (RLN) is an insulin-like hormone associated with extracellular matrix degradation, osteoclastogenesis, and osteoblast differentiation. This study aimed to assess the effect of RLN during and after lateral expansion o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::d96f892965118b21e9091a54d751eeed
https://pubmed.ncbi.nlm.nih.gov/27141932
https://pubmed.ncbi.nlm.nih.gov/27141932
Autor:
Jumpei Morita, Yoshiro Matsui, Toshinori Iwai, Shihomi Shoji, Makoto Adachi, Takafumi Fukui, Kei Watanuki, Kenji Mitsudo, Iwai Tohnai
Publikováno v:
Journal of Japan Society for Oral Tumors. 20:151-156
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0e5508059e18e2d53fc9e7b0cc417613
https://doi.org/10.5843/jsot.20.151
https://doi.org/10.5843/jsot.20.151
Autor:
Yoshihiro Sasaki, Keiji Moriyama, Kazunari Akiyoshi, Yoshihide Hashimoto, Yukiho Kobayashi, Jumpei Morita, Hiroyuki Suzuki, Masako Yokota
Publikováno v:
PLoS ONE, Vol 9, Iss 7, p e101693 (2014)
PLoS ONE
PLoS ONE
Apert syndrome is an autosomal dominantly inherited disorder caused by missense mutations in fibroblast growth factor receptor 2 (FGFR2). Surgical procedures are frequently required to reduce morphological and functional defects in patients with Aper
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9c64473da85f4a2d1d98be4231ee3696
http://europepmc.org/articles/PMC4086955?pdf=render
http://europepmc.org/articles/PMC4086955?pdf=render
Autor:
Chu-Xia Deng, Masataka Nakamura, Keiji Moriyama, Jumpei Morita, Noriko Funato, Yukiho Kobayashi
Publikováno v:
Developmental dynamics : an official publication of the American Association of Anatomists. 243(4)
Background: Apert syndrome (AS) is characterized by craniosynostosis, midfacial hypoplasia, and bony syndactyly. It is an autosomal dominantly inherited disease caused by point mutations (S252W or P253R) in fibroblast growth factor receptor (FGFR) 2.
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::0b2cdda28e088c37e4b58b0246200ec2
https://pubmed.ncbi.nlm.nih.gov/24259495
https://pubmed.ncbi.nlm.nih.gov/24259495
Publikováno v:
Bioorganicmedicinal chemistry letters. 19(13)
1,N(6)-Ethenoadenosine derivatives have been applied as fluorescence probes in various fields of biochemistry and molecular biology. We developed a 1,N(6)-ethenoadenosine-forming reaction at a target adenine in DNA duplex and applied it to a mutation
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::90d005a8087f4e0de2c69a3d98c1cb2c
https://pubmed.ncbi.nlm.nih.gov/19464174
https://pubmed.ncbi.nlm.nih.gov/19464174
Publikováno v:
European Journal of Orthodontics; Jun2017, Vol. 39 Issue 3, p227-234, 8p