Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Julie Magusto"'
Autor:
Paula Ortega-Prieto, Lucia Parlati, Fadila Benhamed, Marion Regnier, Isadora Cavalcante, Mélanie Montabord, Rachel Onifarasoaniaina, Maryline Favier, Natasa Pavlovic, Julie Magusto, Michèle Cauzac, Patrick Pagesy, Jérémie Gautheron, Chantal Desdouets, Sandra Guilmeau, Tarik Issad, Catherine Postic
Publikováno v:
JHEP Reports, Vol 6, Iss 6, Pp 101076- (2024)
Externí odkaz:
https://doaj.org/article/9628daeea97646db92182c02acc0c9bb
Autor:
Paula Ortega-Prieto, Lucia Parlati, Fadila Benhamed, Marion Regnier, Isadora Cavalcante, Mélanie Montabord, Rachel Onifarasoaniaina, Maryline Favier, Natasa Pavlovic, Julie Magusto, Michèle Cauzac, Patrick Pagesy, Jérémie Gautheron, Chantal Desdouets, Sandra Guilmeau, Tarik Issad, Catherine Postic
Publikováno v:
JHEP Reports, Vol 6, Iss 2, Pp 100878- (2024)
Background & Aims: O-GlcNAcylation is a reversible post-translational modification controlled by the activity of two enzymes, O-GlcNAc transferase (OGT) and O-GlcNAcase (OGA). In the liver, O-GlcNAcylation has emerged as an important regulatory mecha
Externí odkaz:
https://doaj.org/article/6bfa0a4c47ff4e52b71882bb7a2bdb3e
Autor:
Julie Magusto, Carine Beaupère, Marta B. Afonso, Martine Auclair, Jean-Louis Delaunay, Pierre-Antoine Soret, Gilles Courtois, Tounsia Aït-Slimane, Chantal Housset, Isabelle Jéru, Bruno Fève, Vlad Ratziu, Cecilia M.P. Rodrigues, Jérémie Gautheron
Publikováno v:
iScience, Vol 25, Iss 10, Pp 105166- (2022)
Summary: Receptor-interacting protein kinase-3 (RIPK3) and mixed lineage kinase domain-like (MLKL) proteins are key regulators of necroptosis, a highly pro-inflammatory mode of cell death, which has been involved in various human diseases. Necroptoti
Externí odkaz:
https://doaj.org/article/144dbd8f28cf4b3c9179202e0635b2cb
Publikováno v:
Biologie Aujourd’hui. 214:1-13
La mort hépatocellulaire chronique et l’inflammation qui en résulte sont des évènements clés dans la progression de la stéatose hépatique non alcoolique (NAFL) vers la stéatohépatite non alcoolique (NASH). La NASH est un état sévère de
Autor:
Marta B. Afonso, Tawhidul Islam, Julie Magusto, Ricardo Amorim, Véronique Lenoir, Rui F. Simões, José Teixeira, Liana C. Silva, Dominique Wendum, Isabelle Jéru, Corinne Vigouroux, Rui E. Castro, Paulo J. Oliveira, Carina Prip‐Buus, Vlad Ratziu, Jérémie Gautheron, Cecília M. P. Rodrigues
Publikováno v:
Hepatology
Hepatology, 2022, ⟨10.1002/hep.32756⟩
Hepatology, 2022, ⟨10.1002/hep.32756⟩
International audience; Background and aims: Receptor-interacting protein kinase 3 (RIPK3) mediates NAFLD progression, but its metabolic function is unclear. Here, we aimed to investigate the role of RIPK3 in modulating mitochondria function, coupled
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::493ae232d3c72a04d1742cbb9981f75f
https://hal.science/hal-03873805/document
https://hal.science/hal-03873805/document
Publikováno v:
Journal of Clinical Medicine
Journal of Clinical Medicine, MDPI, 2020, 10 (1), pp.36. ⟨10.3390/jcm10010036⟩
Journal of Clinical Medicine, 2020, 10 (1), pp.36. ⟨10.3390/jcm10010036⟩
Journal of Clinical Medicine, Vol 10, Iss 36, p 36 (2021)
Journal of Clinical Medicine, MDPI, 2020, 10 (1), pp.36. ⟨10.3390/jcm10010036⟩
Journal of Clinical Medicine, 2020, 10 (1), pp.36. ⟨10.3390/jcm10010036⟩
Journal of Clinical Medicine, Vol 10, Iss 36, p 36 (2021)
International audience; Non-alcoholic fatty liver disease (NAFLD), including non-alcoholic fatty liver (NAFL) and non-alcoholic steatohepatitis (NASH), represents the hepatic manifestation of obesity and metabolic syndrome. Due to the spread of the o
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::67f5df186ea434ef9984b734261c5029
https://hal.sorbonne-universite.fr/hal-03146351/document
https://hal.sorbonne-universite.fr/hal-03146351/document
Publikováno v:
Biologie aujourd'hui. 214(1-2)
Continuous cell death associated with inflammation is a key trigger of disease progression notably in chronic liver diseases such as non-alcoholic steatohepatitis (NASH). Apoptosis has been studied as a potential target for reducing cell death in NAS