Zobrazeno 1 - 10
of 12
pro vyhledávání: '"Julie M. Quach"'
Autor:
Jacqueline E. Noll, Sharon A. Williams, Christine M. Tong, Hongsheng Wang, Julie M. Quach, Louise E. Purton, Katherine Pilkington, Luen B. To, Andreas Evdokiou, Stan Gronthos, Andrew C.W. Zannettino
Publikováno v:
Haematologica, Vol 99, Iss 1 (2014)
Multiple myeloma is an incurable hematologic cancer characterized by the clonal proliferation of malignant plasma cells within the bone marrow. Numerous studies suggest that the myeloma plasma cells occupy and alter the stromal tissue of the bone mar
Externí odkaz:
https://doaj.org/article/f943a3eec82b4af0aaf22185ba44443d
Autor:
Lenny Straszkowski, Alistair M. Chalk, Julie M. Quach, Louise E. Purton, Samuel C. Lee, Takaharu Kimura, Joy Y. Wu, Diannita Kwang, Gavin Tjin, Alanna C. Green, Emma Baker
Publikováno v:
Blood. 138:304-317
Hematopoiesis is extrinsically controlled by cells of the bone marrow microenvironment, including skeletal lineage cells. The identification and subsequent studies of distinct subpopulations of maturing skeletal cells is currently limited because of
Autor:
Nicholas C Wong, Lee H Wong, Julie M Quach, Paul Canham, Jeffrey M Craig, Jenny Z Song, Susan J Clark, K H Andy Choo
Publikováno v:
PLoS Genetics, Vol 2, Iss 2, p e17 (2006)
DNA methylation is a hallmark of transcriptional silencing, yet transcription has been reported at the centromere. To address this apparent paradox, we employed a fully sequence-defined ectopic human centromere (or neocentromere) to investigate the r
Externí odkaz:
https://doaj.org/article/364a206b02114fc7b4469d9f238ce8a7
Autor:
Cristina Lo Celso, Chacko Joseph, Julie M. Quach, Carl R. Walkley, Steven W. Lane, Louise E. Purton
Publikováno v:
Cell Stem Cell. 13:520-533
In recent years, technical developments in mouse genetics and imaging equipment have substantially advanced our understanding of hematopoietic stem cells (HSCs) and their niche. The availability of numerous Cre strains for targeting HSCs and microenv
Autor:
Christine M. Tong, Katherine R. Pilkington, Stan Gronthos, Sharon A. Williams, L. B. To, Louise E. Purton, Jacqueline E. Noll, Hongsheng Wang, Julie M. Quach, Andreas Evdokiou, Andrew C.W. Zannettino
Publikováno v:
Haematologica. 99:163-171
Multiple myeloma is an incurable haematological cancer characterised by the clonal proliferation of malignant plasma cells within the bone marrow. Numerous studies suggest that the myeloma plasma cells occupy and alter the stromal tissue of the bone
Autor:
Julie M. Quach, T. John Martin, Patricia W. M. Ho, Emma C. Walker, Ingrid J Poulton, Narelle E. McGregor, Natalie A. Sims, E H Allan
Publikováno v:
Journal of Bone and Mineral Research. 27:902-912
Parathyroid hormone (PTH) is the only approved anabolic agent for osteoporosis treatment. It acts via osteoblasts to stimulate both osteoclast formation and bone formation, with the balance between these two activities determined by the mode of admin
Autor:
Carl R. Walkley, Kirby E White, Julie M. Quach, Hannah A King, Emma Baker, Maria Askmyr, Tanja Jovic, Louise E. Purton, Ana C Maluenda, Monique Smeets
Publikováno v:
Scientific Reports
Scientific Reports; 5, no 15529 (2015)
Scientific Reports; 5, no 15529 (2015)
The gp130 receptor and its binding partners play a central role in cytokine signalling. Ciliary neurotrophic factor (CNTF) is one of the cytokines that signals through the gp130 receptor complex. CNTF has previously been shown to be a negative regula
Autor:
Peter R. Ebeling, Julie M. Quach, Nicole C. Walsh, Tanja Jovic, David Ritchie, Emma Baker, Simon J. Harrison, Louise E. Purton, Maria Askmyr, Paul J Neeson
Publikováno v:
Journal of bone and mineral research : the official journal of the American Society for Bone and Mineral Research. 30(5)
Skeletal-related events resulting from accelerated bone loss are common complications in patients treated for a range of cancers. However, the mechanisms and rate of bone loss after myelosuppression are unclear. We, therefore, investigated this in mi
Autor:
Elisabetta DeLuca, Joerg Heierhorst, Julie M. Quach, Andrew J. Deans, Carl R. Walkley, Louise E. Purton, Alistair M. Chalk, David J. Izon, Monique Smeets, Meaghan Wall
Mutations within the gene encoding the DNA helicase RECQL4 underlie the autosomal recessive cancer-predisposition disorder Rothmund-Thomson syndrome, though it is unclear how these mutations lead to disease. Here, we demonstrated that somatic deletio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::171cc88c5cb2e519a7ba65c8970b69a9
https://acuresearchbank.acu.edu.au/item/86z73/the-rothmund-thomson-syndrome-helicase-recql4-is-essential-for-hematopoiesis
https://acuresearchbank.acu.edu.au/item/86z73/the-rothmund-thomson-syndrome-helicase-recql4-is-essential-for-hematopoiesis
Autor:
Julie M. Quach, Natalie A. Sims, Atsushi Yokoyama, T. John Martin, Emma C. Walker, Matthew T. Gillespie, Shigeaki Kato, Melissa Solano, E H Allan
Publikováno v:
The Journal of biological chemistry. 286(6)
Osteoblasts and adipocytes are derived from common mesenchymal progenitor cells. The bone loss of osteoporosis is associated with altered progenitor differentiation from an osteoblastic to an adipocytic lineage. cDNA microarrays and quantitative real