Zobrazeno 1 - 7
of 7
pro vyhledávání: '"Julie Bex-Coudrat"'
Autor:
John TCHEN, Quentin SIMON, Léa CHAPART, Morgane K. THAMINY, Shamila VIBHUSHAN, Loredana SAVEANU, Yasmine LAMRI, Fanny SAIDOUNE, Emeline PACREAU, Christophe PELLEFIGUES, Julie BEX-COUDRAT, Hajime KARASUYAMA, Kensuke MIYAKE, Juan HIDALGO, Padraic G. FALLON, Thomas PAPO, Ulrich BLANK, Marc BENHAMOU, Guillaume HANOUNA, Karim SACRE, Eric DAUGAS, Nicolas CHARLES
Publikováno v:
Nature Communications, Vol 15, Iss 1, Pp 1-18 (2024)
Abstract Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by anti-nuclear autoantibodies whose production is promoted by autoreactive T follicular helper (TFH) cells. During SLE pathogenesis, basophils accumulate in secondary
Externí odkaz:
https://doaj.org/article/2b261c12033c4401a2b53ad4f143bfa7
Autor:
Georges Deschênes, Marion Rabant, Laureline Berthelot, Lilia Abbad, Fanny Canesi, Tim Ulinski, Jennifer Da Silva, Michel Peuchmaur, Patrick J. Gleeson, Renato C. Monteiro, Julien Hogan, Alexandra Cambier, Julie Bex-Coudrat, Olivia Boyer
Publikováno v:
Kidney International. 101:274-287
Childhood IgA nephropathy (IgAN) includes a wide spectrum of clinical presentations, from isolated hematuria to acute nephritis with rapid loss of kidney function. In adults, IgAN is an autoimmune disease and its pathogenesis involves galactose defic
Autor:
John Tchen, Quentin Simon, Léa Chapart, Yasmine Lamri, Fanny Saidoune, Emeline Pacreau, Christophe Pellefigues, Julie Bex-Coudrat, Hajime Karasuyama, Kensuke Miyake, Juan Hidalgo, Padraic G. Fallon, Thomas Papo, Ulrich Blank, Marc Benhamou, Guillaume Hanouna, Karim Sacre, Eric Daugas, Nicolas Charles
Systemic lupus erythematosus (SLE) is an autoimmune disease characterized by autoantibodies raised against nuclear antigens and whose production is promoted by autoreactive T follicular helper (TFH) cells. Basophils, by accumulating in secondary lymp
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::89859d8d660a5240c4dd357d58cd075a
https://doi.org/10.1101/2023.01.10.23284399
https://doi.org/10.1101/2023.01.10.23284399
Autor:
Iris K. Madera-Salcedo, Eric Daugas, Emeline Pacreau, Luca Danelli, Maguelonne Pons, Julie Bex-Coudrat, Lydia Celia Madjene, Loïc Rolas, Celine Vaugier, Marc Benhamou, Gunnar Pejler, Walid Beghdadi, Ulrich Blank, Alaa El Ghoneimi, Ivan C. Moura, Julien Claver, Nicolas Charles, Magnus Åbrink, Axel Périanin, Albert Dahdah, Pierre Launay, Shamila Vibhushan
Publikováno v:
Kidney International. 97:516-527
Here we investigated the role of murine mast cell protease 4 (MCPT4), the functional counterpart of human mast cell chymase, in an experimental model of renal ischemia reperfusion injury, a major cause of acute kidney injury. MCPT4-deficient mice had
Autor:
Jonathan M. Chemouny, Erwan Boedec, Karine Le Roux, Gabriella Lauriero, Céline Monot, Marion Leclerc, Julie Bex-Coudrat, Laureline Berthelot, Loreto Gesualdo, Patricia Lepage, Maxime Bredel, Patrick J. Gleeson, Sanae Ben Mkaddem, François Vrtovsnik, Aurélie Sannier, Renato C. Monteiro, Eric Daugas, Lilia Abbad
Publikováno v:
Nephrology Dialysis Transplantation
Nephrology Dialysis Transplantation, Oxford University Press (OUP), 2019, 34 (7), pp.1135-1144. ⟨10.1093/ndt/gfy323⟩
Nephrology Dialysis Transplantation, Oxford University Press (OUP), 2019, 34 (7), pp.1135-1144. ⟨10.1093/ndt/gfy323⟩
Background Immunoglobulin A nephropathy (IgAN) is the most common primary glomerulonephritis worldwide. IgA is mainly produced by the gut-associated lymphoid tissue (GALT). Both experimental and clinical data suggest a role of the gut microbiota in t
Autor:
Agnès Jamin, Lilia Abbad, Christelle Moal, Paolo Martini, Christina Papista, Laureline Berthelot, Julie Bex-Coudrat, Marie-Bénédicte Le Stang, Julien Maillard, Erwan Boedec, Yong Wang, Renato C. Monteiro, Aiqun Li, Sebastian M. Lechner
Publikováno v:
Journal of the American Society of Nephrology : JASN. 27(9)
IgA nephropathy (IgAN), characterized by mesangial IgA1 deposits, is a leading cause of renal failure worldwide. IgAN pathogenesis involves circulating hypogalactosylated IgA1 complexed with soluble IgA Fc receptor I (sCD89) and/or anti-hypogalactosy
Autor:
Martin Flamant, Marie-Bénédicte LeStang, Minas Yiangou, Laureline Berthelot, Mathieu Jablonski, Sanae Ben Mkaddem, François Vrtovsnik, Julie Bex-Coudrat, Jonathan M. Chemouny, Lilia Abbad, Christina Papista, Sebastian M. Lechner, Renato C. Monteiro, Eric Daugas, Evangeline Pillebout
Publikováno v:
Kidney international. 88(2)
IgA1 complexes containing deglycosylated IgA1, IgG autoantibodies, and a soluble form of the IgA receptor (sCD89), are hallmarks of IgA nephropathy (IgAN). Food antigens, notably gluten, are associated with increased mucosal response and IgAN onset,