Zobrazeno 1 - 6
of 6
pro vyhledávání: '"Julianne Elvenes"'
Publikováno v:
PLoS ONE, Vol 9, Iss 1, p e85262 (2014)
Organisms exposed to oxidative stress respond by orchestrating a stress response to prevent further damage. Intracellular levels of antioxidant agents increase, and damaged components are removed by autophagy induction. The KEAP1-NRF2 signaling pathw
Externí odkaz:
https://doaj.org/article/83fa36f08a33453c8c5f50c7009846aa
Autor:
Julianne Elvenes, Ernst Ivan Simon Thomassen, Sylvia Sagen Johnsen, Katrine Kaino, Eva Sjøttem, Terje Johansen
Publikováno v:
PLoS ONE, Vol 6, Iss 9, p e24659 (2011)
The androgen receptor (AR) has a central role in development and maintenance of the male reproductive system and in the etiology of prostate cancer. The transcription factor Pax6 has recently been reported to act as a repressor of AR and to be hyperm
Externí odkaz:
https://doaj.org/article/7cbd8816b9f6472c935735fd4376c449
Publikováno v:
Rheumatology. 57:2246-2248
Autor:
Terje Johansen, Eva Sjøttem, Tor Erik Rusten, Jack-Ansgar Bruun, Nadja Sandra Katheder, Trond Lamark, Julianne Elvenes, Ashish Jain
Publikováno v:
Journal of Biological Chemistry. 290:14945-14962
The selective autophagy receptor p62/sequestosome 1 (SQSTM1) interacts directly with LC3 and is involved in oxidative stress signaling in two ways in mammals. First, p62 is transcriptionally induced upon oxidative stress by the NF-E2-related factor 2
Publikováno v:
Cellular and Molecular Life Sciences. 67:4079-4094
The transcription factor Pax6 is crucial for the embryogenesis of multiple organs, including the eyes, parts of the brain and the pancreas. Mutations in one allele of PAX6 lead to eye diseases including Peter's anomaly and aniridia. Here, we use fluo
Publikováno v:
PLoS ONE
PLoS ONE, Vol 9, Iss 1, p e85262 (2014)
PLoS ONE, Vol 9, Iss 1, p e85262 (2014)
Organisms exposed to oxidative stress respond by orchestrating a stress response to prevent further damage. Intracellular levels of antioxidant agents increase, and damaged components are removed by autophagy induction. The KEAP1-NRF2 signaling pathw