Zobrazeno 1 - 10
of 13
pro vyhledávání: '"Juliana Calle Isaza"'
Publikováno v:
Iatreia, Vol 27, Iss 3, Pp 309-319 (2014)
Autoimmune blistering diseases of the pemphigus group Blisters may appear in many dermatological diseases, but they are not necessarily of autoimmune etiology. For the study of blistering diseases, it is necessary to take into account the clinical as
Externí odkaz:
https://doaj.org/article/d833f9b7f70e4afda0aed4f73c0492e4
Publikováno v:
Nasza Dermatologia Online, Vol 4, Iss 4, Pp 476-478 (2013)
Introduction: The in situ immune response within skin biopsies from patients affected by autoimmune skin blistering diseases (ABDs) is not well characterized. Aim: Based on the fact that the ABD immune response is considered an adaptive immune respon
Externí odkaz:
https://doaj.org/article/0ee83657596f47a3b3cca3021b84f992
Publikováno v:
Nasza Dermatologia Online, Vol 5, Iss 2, Pp 113-117 (2014)
Introduction: Previous research on autoimmune skin blistering diseases (ABD) has primarily focused on the humoral immune response; moreover, little attention has been given to the potential role of the antigen presenting cells (APCs) in lesional skin
Publikováno v:
Nasza Dermatologia Online, Vol 4, Iss suppl.3, Pp 621-624 (2013)
Introduction: Bullous pemphigoid (BP) is one of the most prevalent autoimmune blistering diseases, and believed to be mediated by autoantibodies and complement. The disorder is categorized by the development of urticarial plaques surmounted by subepi
Autor:
Natalia Regina Mesa-Herrera, Jorge Enrique, Hong Yi, Juliana Calle-Isaza, Velásquez Velez, Georgia Dermatopathology, Paul B. Googe, Yulieth Alexandra Upegui, Isabel Cristina Avila, Ana Maria Abreu Velez, Michael S. Howard, Maria Mercedes Yepes Naranjo, Martha Luz Londoño
Publikováno v:
Nasza Dermatologia Online, Vol 4, Iss 3, Pp 275-280 (2013)
Introduction: Proteinases and proteinase inhibitors have been described to play a role in autoimmune skin blistering diseases. We studied skin lesional biopsies from patients affected by several autoimmune skin blistering diseases for proteinases and
Publikováno v:
Repositorio UdeA
Universidad de Antioquia
instacron:Universidad de Antioquia
Redalyc
Universidad de Antioquia
instacron:Universidad de Antioquia
Redalyc
RESUMEN: En muchas enfermedades dermatológicas se presentan ampollas, pero no todas son de etiología autoinmune. Para el estudio de las enfermedades ampollosas se deben tener en cuenta las manifestaciones clínicas, la historia de cómo y cuándo e
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9706eab5106d72ee21bd83dc3ce24a73
Publikováno v:
North American Journal of Medical Sciences
Background: Autoimmune vesiculobullous disorders represent a heterogeneous group of dermatoses whose diagnosis is made based on clinical history, histologic features, and immunopathologic features. The most commonly used techniques for the diagnosis
Publikováno v:
Nasza Dermatologia Online, Vol 5, Iss 2, Pp 125-128 (2014)
Introduction: Human genes responsible for human antigen presentation and transplant rejection functions are located on the short arm of Chromosome 6 and are called the Major Histocompatibility Complex (MHC). Moreover, the primary physiologic function
Externí odkaz:
https://doaj.org/article/934ad9ec7c03483f8ef4f20274c2ec15
Publikováno v:
Nasza Dermatologia Online, Vol 5, Iss 2, Pp 113-117 (2014)
Introduction: Previous research on autoimmune skin blistering diseases (ABD) has primarily focused on the humoral immune response; moreover, little attention has been given to the potential role of the antigen presenting cells (APCs) in lesional skin
Externí odkaz:
https://doaj.org/article/3e9ec52b30da432589d684dcdee23e82
Publikováno v:
Nasza Dermatologia Online, Vol 4, Iss suppl.3, Pp 621-624 (2013)
Introduction: Bullous pemphigoid (BP) is one of the most prevalent autoimmune blistering diseases, and believed to be mediated by autoantibodies and complement. The disorder is categorized by the development of urticarial plaques surmounted by subepi
Externí odkaz:
https://doaj.org/article/dcd31f03e40442b094b7944dceacf9c9