Zobrazeno 1 - 10 of 16 pro vyhledávání: '"Julia Svensson"'
1
Akademický článek
Development of a proteomic signature associated with severe disease for patients with COVID-19 using data from 5 multicenter, randomized, controlled, and prospective studies
Autor: Sandra Castro-Pearson, Sarah Samorodnitsky, Kaifeng Yang, Sahar Lotfi-Emran, Nicholas E. Ingraham, Carolyn Bramante, Emma K. Jones, Sarah Greising, Meng Yu, Brian T. Steffen, Julia Svensson, Eric Åhlberg, Björn Österberg, David Wacker, Weihua Guan, Michael Puskarich, Anna Smed-Sörensen, Elizabeth Lusczek, Sandra E. Safo, Christopher J. Tignanelli
Publikováno v: Scientific Reports, Vol 13, Iss 1, Pp 1-14 (2023)
Abstract Significant progress has been made in preventing severe COVID-19 disease through the development of vaccines. However, we still lack a validated baseline predictive biologic signature for the development of more severe disease in both outpat
Externí odkaz: https://doaj.org/article/8c6ba0735d984f7a80f031a5f8cacf53
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3
Akademický článek
Delayed generation of functional virus-specific circulating T follicular helper cells correlates with severe COVID-19
Autor: Meng Yu, Afandi Charles, Alberto Cagigi, Wanda Christ, Björn Österberg, Sara Falck-Jones, Lida Azizmohammadi, Eric Åhlberg, Ryan Falck-Jones, Julia Svensson, Mu Nie, Anna Warnqvist, Fredrika Hellgren, Klara Lenart, Rodrigo Arcoverde Cerveira, Sebastian Ols, Gustaf Lindgren, Ang Lin, Holden Maecker, Max Bell, Niclas Johansson, Jan Albert, Christopher Sundling, Paulo Czarnewski, Jonas Klingström, Anna Färnert, Karin Loré, Anna Smed-Sörensen
Publikováno v: Nature Communications, Vol 14, Iss 1, Pp 1-14 (2023)
Abstract Effective humoral immune responses require well-orchestrated B and T follicular helper (Tfh) cell interactions. Whether these interactions are impaired and associated with COVID-19 disease severity is unclear. Here, longitudinal blood sample
Externí odkaz: https://doaj.org/article/5d37811980464becbf0604481a523bb4
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4
Akademický článek
Molecular Chaperone BRICHOS Inhibits CADASIL-Mutated NOTCH3 Aggregation In Vitro
Autor: Daniel V. Oliveira, Julia Svensson, Xueying Zhong, Henrik Biverstål, Gefei Chen, Helena Karlström
Publikováno v: Frontiers in Molecular Biosciences, Vol 9 (2022)
CADASIL (cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy) is the most common familial form of stroke, which is caused by mutations located in the epidermal growth factor (EGF)-like repeats of the NOTCH3 gene
Externí odkaz: https://doaj.org/article/c752efbc3c43496cb2da3ed32b323270
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5
Akademický článek
Airway antibodies emerge according to COVID-19 severity and wane rapidly but reappear after SARS-CoV-2 vaccination
Autor: Alberto Cagigi, Meng Yu, Björn Österberg, Julia Svensson, Sara Falck-Jones, Sindhu Vangeti, Eric Åhlberg, Lida Azizmohammadi, Anna Warnqvist, Ryan Falck-Jones, Pia C. Gubisch, Mert Ödemis, Farangies Ghafoor, Mona Eisele, Klara Lenart, Max Bell, Niclas Johansson, Jan Albert, Jörgen Sälde, Deleah D. Pettie, Michael P. Murphy, Lauren Carter, Neil P. King, Sebastian Ols, Johan Normark, Clas Ahlm, Mattias N. Forsell, Anna Färnert, Karin Loré, Anna Smed-Sörensen
Publikováno v: JCI Insight, Vol 6, Iss 22 (2021)
Understanding the presence and durability of antibodies against SARS-CoV-2 in the airways is required to provide insights into the ability of individuals to neutralize the virus locally and prevent viral spread. Here, we longitudinally assessed both
Externí odkaz: https://doaj.org/article/503f709ddbbf4c38ad21dcd3e7009aba
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9
Molecular Chaperone BRICHOS Inhibits CADASIL-Mutated NOTCH3 Aggregation
Autor: Daniel V, Oliveira, Julia, Svensson, Xueying, Zhong, Henrik, Biverstål, Gefei, Chen, Helena, Karlström
Publikováno v: Frontiers in molecular biosciences. 9
CADASIL (cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy) is the most common familial form of stroke, which is caused by mutations located in the epidermal growth factor (EGF)-like repeats of the
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=pmid________::8ee725a14cb5af0fbe192b28299fc79d
https://pubmed.ncbi.nlm.nih.gov/35223989
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