Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Julia Stindl"'
Autor:
Rene Barro-Soria, Julia Stindl, Claudia Müller, Renate Foeckler, Vladimir Todorov, Hayo Castrop, Olaf Strauß
Publikováno v:
PLoS ONE, Vol 7, Iss 11, p e49624 (2012)
Angiotensin II (AngII) receptor (ATR) is involved in pathologic local events such as neovascularisation and inflammation including in the brain and retina. The retinal pigment epithelium (RPE) expresses ATR in its AT1R form, angiotensin-receptor-asso
Externí odkaz:
https://doaj.org/article/17667dfb345a4f978fad0ef16b202063
Autor:
Nancy Ann Oberheim-Bush, Wenyin Shi, Michael W. McDermott, Alexander Grote, Julia Stindl, Leonardo Lustgarten
Publikováno v:
Journal of neuro-oncology, vol 158, iss 3
Introduction Tumor Treating Fields (TTFields, 200 kHz) therapy is a noninvasive, locoregional cancer treatment approved for use in newly diagnosed glioblastoma (GBM), recurrent GBM, and malignant pleural mesothelioma. GBM patients with hydrocephalus
Feasibility and safety of TTFields therapy for glioblastoma patients with ventriculoperitoneal shunt
Autor:
Nancy Ann Oberheim-Bush, Wenyin Shi, Michael W. McDermott, Alexander Grote, Julia Stindl, Leonardo Lustgarten
Publikováno v:
WFNOS 2022 Abstract Book.
Autor:
Leonardo Lustgarten, Alexander Grote, Julia Stindl, Wenyin Shi, Nancy Ann Oberheim-Bush, Michael McDermott
Publikováno v:
Neuro Oncol
INTRODUCTION Hydrocephalus is a common development in patients with glioblastoma (GBM) requiring treatment with ventriculo-peritoneal (VP)-shunts (programmable/non-programmable). To maximize outcomes in this high-risk population, evaluation of VP-shu
Autor:
Olaf Strauß, Nobert Kociok, Hayo Castrop, Joana Raquel Martins, Nadine Reichhart, Julia Stindl, Vladimir T. Todorov, Renate Foeckler, Peter Lachmann
Publikováno v:
Experimental eye research. 189
Several lines of evidence support the existence of a renin-angiotensin system (RAS) in the retina that is separated from the blood stream by the retinal pigment epithelium (RPE). Under physiological conditions, increased activity of intraretinal RAS
Autor:
Christian Huber, Catharina Busch, Olaf Strauß, Ingmar Jost, Julia Stindl, Andreas Genewsky, Peter F. Zipfel, Christine Skerka, Bärbel Rohrer
Publikováno v:
Pflügers Archiv - European Journal of Physiology. 467:2179-2191
Defective regulation of the alternative pathway of the complement system is believed to contribute to damage of retinal pigment epithelial (RPE) cells in age-related macular degeneration. Thus we investigated the effect of complement activation on th
Autor:
B. Aichinger, C. Christ, Sascha Bandulik, Richard Warth, Philipp Tauber, Y. Rhayem, Julia Stindl, Felix Beuschlein
Publikováno v:
ResearcherID
Adrenal aldosterone-producing adenomas (APAs) are a main cause for primary aldosteronism leading to arterial hypertension. Physiologically, aldosterone production in the adrenal gland is stimulated by angiotensin II and high extracellular potassium.
Autor:
Julia Stindl, Christina Sterner, Philipp Tauber, Ines Tegtmeier, Sascha Bandulik, Richard Warth
Publikováno v:
Endocrinology. 156(12)
Aldosterone-producing adenoma (APA) is a major cause of primary aldosteronism, leading to secondary hypertension. Somatic mutations in the gene for the α1 subunit of the Na(+)/K(+)-ATPase were found in about 6% of APAs. APA-related α1 subunit of th
Autor:
Mihail Zilbermint, Silvia Monticone, Anatoly Tiulpakov, Julia Stindl, Michael Allgaeuer, Sascha Bandulik, Ivan Ivanovich Dedov, Constantine A. Stratakis, Paolo Mulatero, Tracy Ann Williams, Chyi-Chia Richard Lee
Publikováno v:
The Journal of clinical endocrinology and metabolism. 100(1)
Familial hyperaldosteronism type III (FH-III) is a rare and clinically heterogeneous condition, that can display mild as well as severe phenotypes. Point mutations in the KCNJ5 gene, affecting the ion selectivity of the inward rectifier K(+) channel
Publikováno v:
Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology. 40(13)
There is growing interest in anxiolytic and pro-social effects of the neuropeptide oxytocin (OXT), but the underlying intraneuronal mechanisms are largely unknown. Here we examined OXT-mediated anxiolysis in the hypothalamic paraventricular nucleus (