Zobrazeno 1 - 5
of 5
pro vyhledávání: '"Julia Kistner"'
Autor:
Tamara Straub, Julia Nave, Pascal Bouvain, Mohammad Akbarzadeh, Siva Sai Krishna Dasa, Julia Kistner, Zhaoping Ding, Aseel Marzoq, Stefanie Stepanow, Katrin Becker, Julia Hesse, Karl Köhrer, Ulrich Flögel, Mohammad R. Ahmadian, Brent A. French, Jürgen Schrader, Sebastian Temme
Publikováno v:
Scientific Reports, Vol 10, Iss 1, Pp 1-14 (2020)
Abstract After myocardial infarction (MI), epicardial cells reactivate their embryonic program, proliferate and migrate into the damaged tissue to differentiate into fibroblasts, endothelial cells and, if adequately stimulated, to cardiomyocytes. Tar
Externí odkaz:
https://doaj.org/article/926c75b0bc764b2880ee83245f14ba59
Autor:
Alexander Jenke, Mariam Yazdanyar, Shunsuke Miyahara, Agunda Chekhoeva, Moritz Benjamin Immohr, Julia Kistner, Udo Boeken, Artur Lichtenberg, Payam Akhyari
Publikováno v:
Journal of the American Heart Association: Cardiovascular and Cerebrovascular Disease, Vol 10, Iss 6 (2021)
Background Cardiac surgery using cardiopulmonary bypass (CPB) frequently provokes a systemic inflammatory response syndrome, which is triggered by TLR4 (Toll‐like receptor 4) and TNF‐α (tumor necrosis factor α) signaling. Here, we investigated
Externí odkaz:
https://doaj.org/article/a2ba68bd4d1d403699ab1325bb3fca90
Autor:
Shunsuke Miyahara, Alexander Jenke, Mariam Yazdanyar, Julia Kistner, Moritz Benjamin Immohr, Yukiharu Sugimura, Hug Aubin, Hiroyuki Kamiya, Yutaka Okita, Artur Lichtenberg, Payam Akhyari
Publikováno v:
Asian cardiovascularthoracic annals. 30(8)
Background It remains unclear whether the Rho-kinase (ROCK) inhibition in combination with mechanical circulatory support (MCS) had a synergic protective effect on myocardial ischemia (MI)/reperfusion injury in therapeutic strategies for acute myocar
Autor:
Julia Kistner, Mariam Yazdanyar, Artur Lichtenberg, Ann Kathrin Bergmann, Payam Akhyari, Sarah Saradar, Alexander Jenke, Melanie Vera Rötepohl, Agunda Chekhoeva
Publikováno v:
American Journal of Physiology-Heart and Circulatory Physiology. 319:H1123-H1141
Calcific aortic valve disease (CAVD) is characterized by valvular fibrosis and calcification and driven by differentiating valvular interstitial cells (VICs). Expression data from patient biopsies suggest that transforming growth factor (TGF)-β1 is
Autor:
Mariam Yazdanyar, Artur Lichtenberg, Payam Akhyari, Alexander Jenke, Sarah Saradar, Julia Kistner
Publikováno v:
Structural Heart. 3:126