Zobrazeno 1 - 10
of 17
pro vyhledávání: '"Juerg Tschopp"'
Autor:
Gisela Niklaus, Sabine Werner, Christine Munding, Juerg Tschopp, Hans-Dietmar Beer, Papin S, Martin Keller
Publikováno v:
Cell Death & Differentiation. 13:1938-1949
The estrogen-responsive B box protein (EBBP) and Pyrin belong to a family of structurally related proteins. While mutations in the pyrin gene cause an autoinflammatory disease, the biological function of EBBP is unknown. In this study, we identified
Autor:
P. A. Fernandez, Michael Schröter, Jean-Claude Martinou, R. Sadoul, J. D. Becherer, Martin Irmler, A. L. Quiquerez, Masatoshi Maki, I. Martinou, Juerg Tschopp
Publikováno v:
Europe PubMed Central
Sympathetic neurons undergo programmed cell death (PCD) upon deprivation of nerve growth factor (NGF). PCD of neurons is blocked by inhibitors of the interleukin-1beta converting enzyme (ICE)/Ced-3-like cysteine protease, indicating involvement of th
Autor:
Mikhail V. Blagosklonny, Eric H. Baehrecke, John M. Abrams, Richard A. Knight, Boris Zhivotovsky, Marcus E. Peter, Douglas R. Green, Walter Malorni, Gerry Melino, Gabriel Núñez, Stuart A. Lipton, Michael O. Hengartner, Junying Yuan, Wafik S. El-Deiry, Pierre Golstein, Mauro Piacentini, Sharad Kumar, Lorenzo Galluzzi, Peter Vandenabeele, Juerg Tschopp, Guido Kroemer, Emad S. Alnemri
Publikováno v:
Cell Death and Differentiation
Cell Death and Differentiation, 2009, 16 (1), pp.3-11. ⟨10.1038/cdd.2008.150⟩
Cell Death and Differentiation, Nature Publishing Group, 2009, 16 (1), pp.3-11. ⟨10.1038/cdd.2008.150⟩
Cell Death and Differentiation, 2009, 16 (1), pp.3-11. ⟨10.1038/cdd.2008.150⟩
Cell Death and Differentiation, Nature Publishing Group, 2009, 16 (1), pp.3-11. ⟨10.1038/cdd.2008.150⟩
International audience; Different types of cell death are often defined by morphological criteria, without a clear reference to precise biochemical mechanisms. The Nomenclature Committee on Cell Death (NCCD) proposes unified criteria for the definiti
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4bd3e815da49f49f93690fb31a6e5ac7
https://hal.science/hal-00407686
https://hal.science/hal-00407686
Autor:
Emmanuelle Logette, Sophie Janssens, Saskia Lippens, Juerg Tschopp, Manfredo Quadroni, Bastienne Jaccard, Solange Cuenin, Antoine Tinel
Publikováno v:
The EMBO journal. 26(1)
Upon DNA damage, a complex called the PIDDosome is formed and either signals NF-kappaB activation and thus cell survival or alternatively triggers caspase-2 activation and apoptosis. PIDD (p53-induced protein with a death domain) is constitutively pr
Publikováno v:
Cell death and differentiation. 13(9)
Members of the viral Flice/caspase-8 inhibitory protein (v-FLIP) family prevent induction of apoptosis by death receptors through inhibition of the processing and activation of procaspase-8 and -10 at the level of the receptor-associated death-induci
Autor:
Andreas Villunger, Andreas Strasser, Michael Schroeter, David C.S. Huang, Juerg Tschopp, Adriano Fontana, Karl Frei, Kim Newton, Michael Hahne
Publikováno v:
Proceedings of the National Academy of Sciences of the United States of America. 96(26)
Fas activation triggers apoptosis in many cell types. Studies with anti-Fas antibodies have produced conflicting results on Fas signaling, particularly the role of the Bcl-2 family in this process. Comparison between physiological ligand and anti-Fas
Publikováno v:
Pathways for Cytolysis ISBN: 9783642794162
The killing mediated by cytotoxic T lymphocytes (CTLs) and natural killer (NK) cells represents an important mechanism in the immune defense against tumors, virus-infected cells, parasites and other foreign invaders. The deleterious effects observed
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::eb89c0fbfcf249b43c380b1b2c68b9b1
https://doi.org/10.1007/978-3-642-79414-8_1
https://doi.org/10.1007/978-3-642-79414-8_1
Autor:
Sylvie Hertig, Roberto Solari, Juerg Tschopp, J. D. Becherer, A Proudfoot, Martin Irmler, R. Sadoul, H R MacDonald
Publikováno v:
The Journal of experimental medicine, vol. 181, no. 5, pp. 1917-1922
The Journal of Experimental Medicine
The Journal of Experimental Medicine
Apoptosis is critically dependent on the presence of the ced-3 gene in Caenorhabditis elegans, which encodes a protein homologous to the mammalian interleukin (IL)-1 beta-converting enzyme (ICE). Overexpression of ICE or ced-3 promotes apoptosis. Cyt
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::9dcfd87510bf3d4298babfe2dbcd15b8
https://serval.unil.ch/notice/serval:BIB_21038A504A30
https://serval.unil.ch/notice/serval:BIB_21038A504A30
Publikováno v:
Cell Death & Differentiation. 7:754-755
Bcl-2 does not inhibit cell death induced by the physiological Fas ligand: implications for the existence of type I and type II cells
Publikováno v:
Scopus-Elsevier
Non-immune activation of the first component of complement (C1) by the heart mitochondrial inner membrane has been investigated. Cardiolipin, the only strong activator of C1 among phospholipids, is present in large amounts in the heart mitochondrial