Zobrazeno 1 - 10
of 36
pro vyhledávání: '"Juan T. Borda"'
Publikováno v:
Revista de Biología Tropical, Vol 48, Iss 1, Pp 255-260 (2000)
The renal lesions are of special importance in the captive primates. The most commonly pathologies are: pyelonephritis, nephrocalcinosis, glomerulonephritis, congenital malformations, hydronephrosis and functional diseases. We report the histopatholo
Externí odkaz:
https://doaj.org/article/5d9f8b7baa274d778ca7a9c4a87f52a1
Autor:
Monica E. Embers, Stephen W. Barthold, Juan T. Borda, Lisa Bowers, Lara Doyle, Emir Hodzic, Mary B. Jacobs, Nicole R. Hasenkampf, Dale S. Martin, Sukanya Narasimhan, Kathrine M. Phillippi-Falkenstein, Jeanette E. Purcell, Marion S. Ratterree, Mario T. Philipp
Publikováno v:
PLoS ONE, Vol 7, Iss 4 (2012)
Externí odkaz:
https://doaj.org/article/6fe2c3af0ca041d386656b487fde723b
Autor:
Xavier Alvarez, Steven D. Douglas, Elizabeth Buza, Pyone P. Aye, Juan T. Borda, Heather Vinet-Oliphant, Mahesh Mohan, Florin Tuluc, Andrew A. Lackner
Publikováno v:
The American Journal of Pathology. 177:1286-1297
Recent studies suggest a link between neuropsychiatric disorders and HIV/SIV infection. Most evidence indicates that monocytes/macrophages are the primary cell type infected within the CNS and that they contribute to CNS inflammation and neurological
Autor:
Juan T. Borda, Woong-Ki Kim, Chie Sugimoto, Atsuhiko Hasegawa, Binhua Ling, Xavier Alvarez, Andrew A. Lackner, Ronald S. Veazey, Huining Liu, Marcelo J. Kuroda, Ruy M. Ribeiro, Kenneth C. Williams, Heather Vinet-Oliphant
Publikováno v:
Blood. 114:2917-2925
It is widely accepted that destruction of CD4+ T cells and viral load are the primary markers for immunodeficiency in HIV-1–infected humans and in simian immunodeficiency virus (SIV)–infected macaques. However, monocyte/macrophages are also impor
Autor:
Pyone P. Aye, Joel V. Weinstock, Armandina Garza, David J. Tweardy, Juan T. Borda, Melinda D'Souza, Jeffrey K. Griffiths, Andrew A. Lackner, Prema Robinson, Protacio Martin
Publikováno v:
Journal of Medical Primatology. 37:109-115
Background Cryptosporidium infection leads to life-threatening diarrhea in AIDS patients. Pathogenesis of cryptosporidiosis is due to intestinal physiological alterations. We devised an ex-vivo model using ex-vivo Cryptosporidium parvum infection of
Autor:
Andrew A. Lackner, Xavier Alvarez, Mahesh Mohan, Juan T. Borda, Marion S. Ratterree, Bruce A. Bunnell, Kathrine M Phillippi-Falkenstein
Publikováno v:
The American Journal of Pathology. 172:98-111
Globoid cell leukodystrophy, or Krabbe's disease, is a severe disorder of the central and peripheral nervous system caused by the absence of galactocerebrosidase (GALC) activity. Herein, we describe the clinical, neuropathological, histochemical, and
Publikováno v:
The American Journal of Pathology. 171:1952-1965
Gastrointestinal disease and inflammation are common sequelae of human and simian immunodeficiency virus (SIV) infection. Nevertheless, the molecular mechanisms that lead to gastrointestinal dysfunction remain unclear. We investigated regulation of t
Autor:
You Zhou, Karol Sestak, Rohana P. Dassanayake, Gerald E. Duhamel, Gail B. Plauché, Juan T. Borda, Susanne Hinkley, Cynthia J. Stryker
Publikováno v:
Journal of Clinical Microbiology. 43:641-649
An association between certainCampylobacterspecies and enterocolitis in humans and nonhuman primates is well established, but the association between cytolethal distending toxin and disease is incompletely understood. The purpose of the present study
Visualizing Cytokine-Secreting Cells In Situ in the Rhesus Macaque Model of Chronic Gut Inflammation
Publikováno v:
Clinical and Vaccine Immunology. 12:192-197
Cytokine-producing cells in gut-associated lymphoid tissues of rhesus macaques with chronic enterocolitis were studied. The confocal microscopy technique that we developed enables simultaneous in situ visualization of multiple extra- and/or intracell
Autor:
Meredith A. Simon, Pyone P. Aye, Juan T. Borda, Xavier Alvarez, Andrew A. Lackner, Ronald C. Desrosiers, Ivanela Kondova
Publikováno v:
The American Journal of Pathology. 165:2111-2122
SIVmac239/316 is a molecular clone derived from SIVmac239 that differs from the parental virus by nine amino acids in env. This virus, unlike the parental SIVmac239, is able to replicate well in alveolar macrophages in culture. We have not however, o