Zobrazeno 1 - 10
of 11
pro vyhledávání: '"Juan M. Reséndiz-Hernández"'
Autor:
Ramcés Falfán-Valencia, Arun Narayanankutty, Juan M. Reséndiz-Hernández, Gloria Pérez-Rubio, Alejandra Ramírez-Venegas, Karol J. Nava-Quiroz, Nora E. Bautista-Félix, Gilberto Vargas-Alarcón, Manuel D. J. Castillejos-López, Andrés Hernández
Publikováno v:
Journal of Immunology Research, Vol 2018 (2018)
Background. The influenza A H1N1/09 pandemic infected a small number of exposed individuals, which suggests the involvement of genetic factors. There are scarce data available on classical HLA class I association with the influenza A H1N1/09 pandemic
Externí odkaz:
https://doaj.org/article/f97936020b01491da962a337b18004fc
Autor:
Edgar Abarca-Rojano, Juan M. Reséndiz-Hernández, Alma D. Del Angel-Pablo, Rafael Hernández-Zenteno, Carlos H.I. Ramos, Martha Montaño, Alejandra Ramírez-Venegas, Gloria Pérez-Rubio, Ramcés Falfán-Valencia, Enrique Ambrocio-Ortiz
Publikováno v:
Cytokine. 111:364-370
Chronic obstructive pulmonary disease (COPD) is a complex and multifactorial disease involving systemic inflammation. Although certain genetic components have been implicated in the development and progression of this disease, few studies have examin
Autor:
Juan M. Reséndiz-Hernández, Edgar Abarca-Rojano, Ana María Salazar, Rafael Hernández-Zenteno, Alejandra Ramírez-Venegas, Fernando Flores-Trujillo, Ángel Camarena, Gloria Pérez-Rubio, Martha Pérez-Rodríguez, Enrique Ambrocio-Ortiz, Ramcés Falfán-Valencia, Luis Alberto López-Flores, Gandhi F. Pavón-Romero
Publikováno v:
International Journal of Chronic Obstructive Pulmonary Disease. 13:627-637
Background Smoking and smoke from biomass burning (BB) are the main environmental risk factors for COPD. Clinical differences have been described between COPD related to smoking and related to wood smoke, but no studies have shown genetic differences
Publikováno v:
Advances in Clinical and Experimental Medicine. 27:125-133
Asthma and chronic obstructive pulmonary disease (COPD) are widely documented diseases with an inflammatory component. Asthma is a heterogeneous disorder of the airways that involves chronic inflammation, decline of the airway function and tissue rem
Autor:
Ángel Camarena, Gilberto Vargas-Alarcón, Rafael Hernández-Zenteno, Mónica Velázquez-Uncal, Ramcés Falfán-Valencia, Alejandra Ramírez-Venegas, Raúl H Sansores, Juan M. Reséndiz-Hernández, Laura Colín-Barenque
Publikováno v:
International Journal of Chronic Obstructive Pulmonary Disease
Juan Manuel Reséndiz-Hernández,1,2 Raúl H Sansores,3 Rafael de Jesús Hernández-Zenteno,3 Gilberto Vargas-Alarcón,4 Laura Colín-Barenque,5 Mónica Velázquez-Uncal,3 Angel Camarena,1 Alejandra Ramírez-Venegas,3 Ramcés Falfán-Valencia1 1Labor
Autor:
Luis Octavio Jiménez-Valverde, Fernando Flores-Trujillo, Alejandra Ramírez-Venegas, Juan M. Reséndiz-Hernández, Gloria Pérez-Rubio, Raúl H Sansores, Ramcés Falfán-Valencia, Ángel Camarena
Publikováno v:
Archivos de Bronconeumología (English Edition). 51:80-85
Introduction Chronic obstructive pulmonary disease (COPD) is characterized by restricted airflow. The best-documented genetic factor is alpha-1 antitrypsin (AAT). AAT is encoded by the SERPINA1 gene. The PiZ (rs28929474) and PiS (rs17580) variants ar
Autor:
Fernando Flores-Trujillo, Luis Octavio Jiménez-Valverde, Juan M. Reséndiz-Hernández, Ángel Camarena, Raúl H Sansores, Ramcés Falfán-Valencia, Alejandra Ramírez-Venegas, Gloria Pérez-Rubio
Publikováno v:
Archivos de Bronconeumología. 51:80-85
Resumen Introduccion La enfermedad pulmonar obstructiva cronica (EPOC) se caracteriza por dificultad para respirar. El factor genetico mejor documentado es la deficiencia de alfa-1 antitripsina (A1AT). La A1AT esta codificada por el gen SERPINA1. Se
Autor:
Luis M. Teran, Juan M. Reséndiz-Hernández, Gloria Pérez-Rubio, Ángel Camarena, Fernando Ramírez-Jiménez, Ramcés Falfán-Valencia, Gandhi F. Pavón-Romero
Publikováno v:
Biomarkers in medicine. 11(12)
Aim: To evaluate the association of three single nucleotide polymorphisms in TNF and one in LTA in Mexican patients with aspirin-exacerbated respiratory disease (AERD) and the correlation of those single nucleotide polymorphisms with serum levels of
Autor:
Gilberto Vargas-Alarcón, Karol J Nava-Quiroz, Nora E. Bautista-Félix, Manuel Castillejos-López, Alejandra Ramírez-Venegas, Gloria Pérez-Rubio, Juan M. Reséndiz-Hernández, Ramcés Falfán-Valencia, Andrés Hernández, Arun Narayanankutty
Publikováno v:
Journal of Immunology Research
Journal of Immunology Research, Vol 2018 (2018)
Journal of Immunology Research, Vol 2018 (2018)
Background. The influenza A H1N1/09 pandemic infected a small number of exposed individuals, which suggests the involvement of genetic factors. There are scarce data available on classical HLA class I association with the influenza A H1N1/09 pandemic
Publikováno v:
Clinical Biochemistry. 46:566-578
Aspirin exacerbated respiratory disease (AERD) is a distinct clinical entity characterized by eosinophilic rhinosinusitis, asthma and often nasal polyposis. Exposure to aspirin or other nonsteroid anti-inflammatory drugs (NSAIDs) exacerbates bronchos