Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Ju Hee Jun"'
Autor:
Hyang Hee Cho, Siyeon Rhee, Dong Im Cho, Ju Hee Jun, HyoJung Heo, Su Han Cho, Dohyup Kim, Mingqiang Wang, Bo Gyeong Kang, Soo Ji Yoo, Meeyoung Cho, Soo yeon Lim, Jae Yeong Cho, In Seok Jeong, Yong Sook Kim, Youngkeun Ahn
Publikováno v:
Experimental and Molecular Medicine, Vol 56, Iss 9, Pp 2052-2064 (2024)
Abstract The regulatory role of the inhibitor of NF-kB kinase ε (IKKε) in postmyocardial infarction (MI) inflammation remains uncertain. Using an MI mouse model, we examined the cardiac outcomes of IKKε knockout (KO) mice and wild-type mice. We em
Externí odkaz:
https://doaj.org/article/ee2d2e2c60f449deaa5e5bc09fa9885e
Autor:
Mingyu Lee, Yong Sook Kim, Junggeon Park, Goeun Choe, Sanghun Lee, Bo Gyeong Kang, Ju Hee Jun, Yoonmin Shin, Minchul Kim, Youngkeun Ahn, Jae Young Lee
Publikováno v:
Bioactive Materials, Vol 31, Iss , Pp 395-407 (2024)
The infarcted heart undergoes irreversible pathological remodeling after reperfusion involving left ventricle dilation and excessive inflammatory reactions in the infarcted heart, frequently leading to fatal functional damage. Extensive attempts have
Externí odkaz:
https://doaj.org/article/98362e30f7bc453a8bd8e9a4b7a70b63
Autor:
Dong Im Cho, Min Joo Ahn, Hyang Hee Cho, Meeyoung Cho, Ju Hee Jun, Bo Gyeong Kang, Soo Yeon Lim, Soo Ji Yoo, Mi Ra Kim, Hyung-Seok Kim, Su-Jin Lee, Le Thanh Dat, Changho Lee, Yong Sook Kim, Youngkeun Ahn
Publikováno v:
Experimental and Molecular Medicine, Vol 55, Iss 2, Pp 426-442 (2023)
Cardiovascular disease: Stopping and stabilizing atherosclerotic plaques Treatment with a protein that stabilizes existing plaques within blood vessels could help reduce the risk of future cardiovascular events in patients with atherosclerosis. These
Externí odkaz:
https://doaj.org/article/39e89f9d113c4639b20e48c9acdb3920
Autor:
Yong Sook Kim, Hyang Hee Cho, Dong Im Cho, Hye-yun Jeong, Soo yeon Lim, Ju Hee Jun, Mi Ra Kim, Bo Gyeong Kang, Meeyoung Cho, Hye-jin Kang, Wan Seok Kang, Goo Taeg Oh, Youngkeun Ahn
Publikováno v:
Cell Death and Disease, Vol 12, Iss 4, Pp 1-15 (2021)
Abstract Resistin-like alpha (Retnla) is a member of the resistin family and known to modulate fibrosis and inflammation. Here, we investigated the role of Retnla in the cardiac injury model. Myocardial infarction (MI) was induced in wild type (WT),
Externí odkaz:
https://doaj.org/article/df6b1926c603404e8490ed4b87d00752
Autor:
Yong Sook Kim, Mira Kim, Dong Im Cho, Soo Yeon Lim, Ju Hee Jun, Mi Ra Kim, Bo Gyeong Kang, Gwang Hyeon Eom, Gaeun Kang, Somy Yoon, Youngkeun Ahn
Publikováno v:
Pharmaceutics, Vol 14, Iss 8, p 1659 (2022)
Intensive research has focused on minimizing the infarct area and stimulating endogenous regeneration after myocardial infarction. Our group previously elucidated that apicidin, a histone deacetylase (HDAC) inhibitor, robustly accelerates the cardiac
Externí odkaz:
https://doaj.org/article/0cf339f8e6404a4faff4e0bc7c2b714e
Autor:
Wan Seok Kang, Meeyoung Cho, Hye-jin Kang, Goo Taeg Oh, Mi Ra Kim, Hyang Hee Cho, Bo Gyeong Kang, Soo Yeon Lim, Youngkeun Ahn, Ju Hee Jun, Yong Sook Kim, Hye yun Jeong, Dong Im Cho
Publikováno v:
Cell Death and Disease, Vol 12, Iss 4, Pp 1-15 (2021)
Cell Death & Disease
Cell Death & Disease
Resistin-like alpha (Retnla) is a member of the resistin family and known to modulate fibrosis and inflammation. Here, we investigated the role of Retnla in the cardiac injury model. Myocardial infarction (MI) was induced in wild type (WT), Retnla kn
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::febeae1332e7b80f367646e85e80e832
https://doaj.org/article/df6b1926c603404e8490ed4b87d00752
https://doaj.org/article/df6b1926c603404e8490ed4b87d00752
Autor:
Min-Young Lim, Han Young Kim, Hye-yun Jeong, Gwang Hyeon Eom, Saerom Kong, Youngkeun Ahn, Ju Hee Jun, Mikyung Kang, Jong-Chan Lee, Jooyeon Park, Byung-Soo Kim, Jin Han, Yong Sook Kim, Seungmi Ryu, Yeon Woong Choo, Gun-Jae Jeong
Publikováno v:
ACS Nano. 12:1959-1977
Development of localized inflammatory environments by M1 macrophages in the cardiac infarction region exacerbates heart failure after myocardial infarction (MI). Therefore, the regulation of inflammation by M1 macrophages and their timely polarizatio
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b7c0540cba2c51435c13137649169d8a
https://doi.org/10.1021/acsnano.7b09107
https://doi.org/10.1021/acsnano.7b09107
Publikováno v:
Circulation Research. 125
Inhibitor of NF-κB kinase (IKK), an upstream of nuclear factor-kappa B (NF-κB), is a critical modulator for pathophysiological inflammation. IKKε is a non-classical IKK and has been studied in infectious diseases and cancers. However, the role of
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::0803ae1ebdece020d363674fa1cf2808
https://doi.org/10.1161/res.125.suppl_1.221
https://doi.org/10.1161/res.125.suppl_1.221