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Autor:
Vincenza Cifarelli, Ondrej Kuda, Kui Yang, Xinping Liu, Richard W. Gross, Terri A. Pietka, Gyu Seong Heo, Deborah Sultan, Hannah Luehmann, Josie Lesser, Morgan Ross, Ira J. Goldberg, Robert J. Gropler, Yongjian Liu, Nada A. Abumrad
Publikováno v:
Frontiers in Cardiovascular Medicine, Vol 9 (2022)
CD36 mediates the uptake of long-chain fatty acids (FAs), a major energy substrate for the myocardium. Under excessive FA supply, CD36 can cause cardiac lipid accumulation and inflammation while its deletion reduces heart FA uptake and lipid content
Externí odkaz:
https://doaj.org/article/bfbdb0257f3749e7ad0b6d0009d78f34