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of 6
pro vyhledávání: '"Joshua C. Ferrell"'
Publikováno v:
PLoS ONE, Vol 9, Iss 4, p e93861 (2014)
The SOS response to DNA damage that induces up to 10% of the prokaryotic genome requires RecA action to relieve LexA transcriptional repression. In Acinetobacter species, which lack LexA, the error-prone polymerase accessory UmuDAb is instead require
Externí odkaz:
https://doaj.org/article/54dcb3577c96470c8f934f7fc8739afd
Publikováno v:
mBio
mBio, Vol 12, Iss 1 (2021)
mBio, Vol 12, Iss 1 (2021)
The increasing genetic tractability of Chlamydia trachomatis is accelerating the ability to characterize the unique infection biology of this obligate intracellular parasite. These efforts are leading to a greater understanding of the molecular event
Autor:
Elizabeth A. Ruelke, Maria Bodero, Susmita Ghosh, Kenneth A. Fields, Travis J. Jewett, Joshua C. Ferrell
Publikováno v:
Infection and Immunity. 88
The translocated actin recruiting phosphoprotein (Tarp) is a multidomain type III secreted effector used by Chlamydia trachomatis In aggregate, existing data suggest a role of this effector in initiating new infections. As new genetic tools began to
Autor:
Joshua C. Ferrell, Kenneth A. Fields
Publikováno v:
Microbes and Infection. 18:84-92
It has been appreciated for almost 20 years that members of the Chlamydiales possess a virulence-associated type III secretion mechanism. Given the obligate intracellular nature of these bacteria, defining exactly how type III secretion functions to
Autor:
Jennifer Hurban, Erin L. Abner, Joshua C. Ferrell, Tiffany L. Sudduth, Elizabeth Head, Tina L. Beckett, Frederick A. Schmitt, M. Paul Murphy, Katie McCarty, Alex M. Helman, Donna M. Wilcock
Publikováno v:
Neurobiology of aging, vol 36, iss 9
Down syndrome (DS) is the most common genetic cause of intellectual disability and is primarily caused by the triplication of chromosome 21. The overexpression of amyloid precursor protein gene may be sufficient to drive Alzheimer's disease (AD) neur
Publikováno v:
PLoS ONE
PLoS ONE, Vol 9, Iss 4, p e93861 (2014)
PLoS ONE, Vol 9, Iss 4, p e93861 (2014)
The SOS response to DNA damage that induces up to 10% of the prokaryotic genome requires RecA action to relieve LexA transcriptional repression. In Acinetobacter species, which lack LexA, the error-prone polymerase accessory UmuDAb is instead require