Zobrazeno 1 - 10 of 13 pro vyhledávání: '"Joshua A. F. Sutton"'
1
Akademický článek
Clonal population expansion of Staphylococcus aureus occurs due to escape from a finite number of intraphagocyte niches
Autor: Grace R. Pidwill, Josie F. Pyrah, Joshua A. F. Sutton, Alex Best, Stephen A. Renshaw, Simon J. Foster
Publikováno v: Scientific Reports, Vol 13, Iss 1, Pp 1-11 (2023)
Abstract Staphylococcus aureus is a human commensal and also an opportunist pathogen causing life threatening infections. During S. aureus disease, the abscesses that characterise infection can be clonal, whereby a large bacterial population is found
Externí odkaz: https://doaj.org/article/be9030a97869411792d99ab1fb4dbab1
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2
Akademický článek
The roles of GpsB and DivIVA in Staphylococcus aureus growth and division
Autor: Joshua A. F. Sutton, Mark Cooke, Mariana Tinajero-Trejo, Katarzyna Wacnik, Bartłomiej Salamaga, Callum Portman-Ross, Victoria A. Lund, Jamie K. Hobbs, Simon J. Foster
Publikováno v: Frontiers in Microbiology, Vol 14 (2023)
The spheroid bacterium Staphylococcus aureus is often used as a model of morphogenesis due to its apparently simple cell cycle. S. aureus has many cell division proteins that are conserved across bacteria alluding to common functions. However, despit
Externí odkaz: https://doaj.org/article/be7c4e711403437dafa287a64898ba25
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3
Akademický článek
Commensal bacteria augment Staphylococcus aureus infection by inactivation of phagocyte-derived reactive oxygen species.
Autor: Josie F Gibson, Grace R Pidwill, Oliver T Carnell, Bas G J Surewaard, Daria Shamarina, Joshua A F Sutton, Charlotte Jeffery, Aurélie Derré-Bobillot, Cristel Archambaud, Matthew K Siggins, Eric J G Pollitt, Simon A Johnston, Pascale Serror, Shiranee Sriskandan, Stephen A Renshaw, Simon J Foster
Publikováno v: PLoS Pathogens, Vol 17, Iss 9, p e1009880 (2021)
Staphylococcus aureus is a human commensal organism and opportunist pathogen, causing potentially fatal disease. The presence of non-pathogenic microflora or their components, at the point of infection, dramatically increases S. aureus pathogenicity,
Externí odkaz: https://doaj.org/article/589ad2747e0c478eacd842d8f79203f3
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4
Akademický článek
Staphylococcus aureus cell wall structure and dynamics during host-pathogen interaction.
Autor: Joshua A F Sutton, Oliver T Carnell, Lucia Lafage, Joe Gray, Jacob Biboy, Josie F Gibson, Eric J G Pollitt, Simone C Tazoll, William Turnbull, Natalia H Hajdamowicz, Bartłomiej Salamaga, Grace R Pidwill, Alison M Condliffe, Stephen A Renshaw, Waldemar Vollmer, Simon J Foster
Publikováno v: PLoS Pathogens, Vol 17, Iss 3, p e1009468 (2021)
Peptidoglycan is the major structural component of the Staphylococcus aureus cell wall, in which it maintains cellular integrity, is the interface with the host, and its synthesis is targeted by some of the most crucial antibiotics developed. Despite
Externí odkaz: https://doaj.org/article/fcebc94d5e2540bd8ac9e1aa6695335c
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5
Akademický článek
Evolving MRSA: High-level β-lactam resistance in Staphylococcus aureus is associated with RNA Polymerase alterations and fine tuning of gene expression.
Autor: Viralkumar V Panchal, Caitlin Griffiths, Hamed Mosaei, Bohdan Bilyk, Joshua A F Sutton, Oliver T Carnell, David P Hornby, Jeffrey Green, Jamie K Hobbs, William L Kelley, Nikolay Zenkin, Simon J Foster
Publikováno v: PLoS Pathogens, Vol 16, Iss 7, p e1008672 (2020)
Most clinical MRSA (methicillin-resistant S. aureus) isolates exhibit low-level β-lactam resistance (oxacillin MIC 2-4 μg/ml) due to the acquisition of a novel penicillin binding protein (PBP2A), encoded by mecA. However, strains can evolve high-le
Externí odkaz: https://doaj.org/article/4eed5d53cf4c4fb0b3f786b85c9adb0f
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7
Antibiotics Limit Adaptation of Drug-Resistant Staphylococcus aureus to Hypoxia
Autor: Rebecca C. Hull, Rosanna C. T. Wright, Jon R. Sayers, Joshua A. F. Sutton, Julia Rzaska, Simon J. Foster, Michael A. Brockhurst, Alison M. Condliffe
Publikováno v: Antimicrobial Agents and Chemotherapy. 66
Bacterial pathogens are confronted with a range of challenges at the site of infection, including exposure to antibiotic treatment and harsh physiological conditions, that can alter the fitness benefits and costs of acquiring antibiotic resistance. H
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::4b3182cc9e3357159be0e84d52681a63
https://doi.org/10.1128/aac.00926-22
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8
Commensal bacteria augment Staphylococcus aureus infection by inactivation of phagocyte-derived reactive oxygen species
Autor: Simon A. Johnston, Eric J. G. Pollitt, Bas G.J. Surewaard, Cristel Archambaud, Pascale Serror, Charlotte Jeffery, Aurélie Derré-Bobillot, Stephen A. Renshaw, Matthew K. Siggins, Grace R. Pidwill, Simon J. Foster, Josie F. Gibson, Joshua A. F. Sutton, Shiranee Sriskandan, Daria Shamarina, Oliver T. Carnell
Publikováno v: PLoS Pathogens
PLoS Pathogens, Public Library of Science, 2021, 17 (9), pp.e1009880. ⟨10.1371/journal.ppat.1009880⟩
PLoS Pathogens, Vol 17, Iss 9, p e1009880 (2021)
Staphylococcus aureus is a human commensal organism and opportunist pathogen, causing potentially fatal disease. The presence of non-pathogenic microflora or their components, at the point of infection, dramatically increases S. aureus pathogenicity,
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::444102e9bb9a168cac86e83e13767fe9
https://hal.archives-ouvertes.fr/hal-03363829
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9
Evolving MRSA: High-level β-lactam resistance in Staphylococcus aureus is associated with RNA Polymerase alterations and fine tuning of gene expression
Autor: Oliver T. Carnell, Nikolay Zenkin, William L. Kelley, Bohdan Bilyk, Jeffrey Green, Viralkumar V. Panchal, Simon J. Foster, Jamie K. Hobbs, Joshua A. F. Sutton, Hamed Mosaei, David P. Hornby, Caitlin Griffiths
Publikováno v: PLoS Pathogens
PLOS Pathogens, Vol. 16, No 7 (2020) P. e1008672
PLoS Pathogens, Vol 16, Iss 7, p e1008672 (2020)
Most clinical MRSA (methicillin-resistant S. aureus) isolates exhibit low-level β-lactam resistance (oxacillin MIC 2–4 μg/ml) due to the acquisition of a novel penicillin binding protein (PBP2A), encoded by mecA. However, strains can evolve high-
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_dedup___::956b4f17bd86062dc65d2eed9ac057c5
http://europepmc.org/articles/PMC7380596
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10
Augmenting Staphylococcal infection: the importance of timing
Autor: Oliver T. Carnell, Simon J. Foster, Joshua A. F. Sutton
Publikováno v: Access Microbiology. 1
Staphylococcus aureus (S. aureus) acts as a commensal in the microbiome of the skin and nasopharynx. However, on gaining access to the bloodstream it can cause an array of pathogenic outcomes. S. aureus can crowdsource the microflora to assist in bec
Externí odkaz: https://explore.openaire.eu/search/publication?articleId=doi_________::c5bb0404dfd97ed66a7fee11f5001e39
https://doi.org/10.1099/acmi.ac2019.po0133
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