Zobrazeno 1 - 10
of 31
pro vyhledávání: '"Josefa González Carrillo"'
Autor:
Josep Ramon Marsal, Laura Gutierrez-Garcia, Juan Ramón Gimeno-Blanes, Pier Giorgio Masci, Juan Jiménez-Jáimez, Gisela Teixido-Tura, Marta Codina-Solà, Gerard Oristrell, Coloma Tiron, Ignacio Ferreira-González, Andrea Guala, Pablo García-Pavía, Paula Fernández-Álvarez, Juan José Santos-Mateo, Esther Zorio, José Luis de la Pompa, Artur Evangelista, José Manuel García-Pinilla, Daniele Andreini, Eduardo Villacorta, Tomás Ripoll-Vera, Ángela López-Sainz, José Rodríguez-Palomares, José Antonio Sorolla-Romero, Gianluca Pontone, Lucia La Mura, Javier Limeres, Jan Bogaert, Mar Borregan, Augusto Sao Avilés, Julián Palomino-Doza, Rafaela Soler-Fernandez, Aida Ribera, Josefa González-Carrillo, José M. Larrañaga-Moreira, Guillem Casas, Giovanni Donato Aquaro, Roberto Barriales-Villa, Antoni Bayes-Genis
Publikováno v:
JOURNAL OF THE AMERICAN COLLEGE OF CARDIOLOGY
r-FSJD. Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
instname
r-FSJD. Repositorio Institucional de Producción Científica de la Fundació Sant Joan de Déu
instname
Left ventricular noncompaction (LVNC) is a heterogeneous entity with uncertain prognosis. This study sought to develop and validate a prediction model of major adverse cardiovascular events (MACE) and to identify LVNC cases without events during long
Publikováno v:
The Journal of Supercomputing. 77:10138-10151
Left ventricular non-compaction (LVNC) is a rare cardiomyopathy characterized by abnormal trabeculations in the left ventricle cavity. Although traditional computer vision approaches exist for LVNC diagnosis, deep learning-based tools could not be fo
Publikováno v:
Journal of Clinical Medicine
Volume 10
Issue 3
Journal of Clinical Medicine, Vol 10, Iss 503, p 503 (2021)
Volume 10
Issue 3
Journal of Clinical Medicine, Vol 10, Iss 503, p 503 (2021)
Left ventricular non-compaction (LVNC) is defined by an increase of trabeculations in left ventricular (LV) endomyocardium. Although LVNC can be in isolation, an increase in hypertrabeculation often accompanies genetic cardiomyopathies. Current metho
Autor:
Marcos Siguero Alvárez, José Luis de la Pompa, Javier Cuenca Muñoz, Gregorio Bernabé García, Angel Raya, Esther Burillo Milla, Juan R. Gimeno, Carmen Muñoz Esparza, José D. Casanova, Jesús Martín Jiménez, Maria Sabater Molina, Rubén Escribá, Josefa González Carrillo
Publikováno v:
Journal of Clinical Medicine
Dipòsit Digital de la UB
Universidad de Barcelona
Repisalud
Instituto de Salud Carlos III (ISCIII)
Volume 9
Issue 10
Journal of Clinical Medicine, Vol 9, Iss 3171, p 3171 (2020)
Dipòsit Digital de la UB
Universidad de Barcelona
Repisalud
Instituto de Salud Carlos III (ISCIII)
Volume 9
Issue 10
Journal of Clinical Medicine, Vol 9, Iss 3171, p 3171 (2020)
Aims: Hypertrophic cardiomyopathy (HCM) is often accompanied by increased trabeculated myocardium (TM)&mdash
which clinical relevance is unknown. We aim to measure the left ventricular (LV) mass and proportion of trabeculation in an HCM populati
which clinical relevance is unknown. We aim to measure the left ventricular (LV) mass and proportion of trabeculation in an HCM populati
Autor:
José M. García, Jesús M. Rodríguez-de-Vera, Josefa González-Carrillo, Daniel Saura, Gregorio Bernabé
Publikováno v:
Computer Methods and Programs in Biomedicine. 214:106548
Background and Objective Left ventricular non-compaction (LVNC) is an uncommon cardiomyopathy characterised by a thick and spongy left ventricle wall caused by the high presence of trabeculae (hyper-trabeculation). Recently, the percentage of the tra
Publikováno v:
The Journal of Supercomputing. 75:1625-1640
Left ventricular non-compaction is characterized by the presence of multiple trabecules in the left ventricle myocardium, associated with multiple inter-trabecular recesses communicated with the ventricular cavity. The medical community needs an obje
Autor:
Domigo Pascual-Figal, Juan R. Gimeno, María Sabater-Molina, Esperanza García-Molina Sáez, Pablo Carbonell, María del Carmen Olmo, Luis Polo, Inmaculada Pérez-Sánchez, María José Oliva-Sandoval, Roberto Barriales-Villa, Daniel Saura, Josefa González-Carrillo
Publikováno v:
Revista Española de Cardiología (English Edition). 70:105-114
Introduction and objectives Mutations in MYBPC3 are the cause of hypertrophic cardiomyopathy (HCM). Although most lead to a truncating protein, the severity of the phenotype differs. We describe the clinical phenotype of a novel MYBPC3 mutation, p.Pr
Autor:
Juan R. Gimeno, Inmaculada Pérez-Sánchez, Roberto Barriales-Villa, Esperanza García-Molina Sáez, María del Carmen Olmo, Josefa González-Carrillo, María Sabater-Molina, Pablo Carbonell, Luis Polo, Daniel Saura, Domigo Pascual-Figal, María José Oliva-Sandoval
Publikováno v:
Revista Española de Cardiología. 70:105-114
Resumen Introduccion y objetivos Las mutaciones en MYBPC3 son causa de miocardiopatia hipertrofica (MCH). A pesar de que la mayoria de ellas producen una proteina truncada, la gravedad del fenotipo es diversa. Se describe el fenotipo clinico de una n
Autor:
Mariano Valdés, Miguel García-Navarro, Patrizio Lancellotti, Josefa González-Carrillo, María José Oliva-Sandoval, Luis Caballero, Daniel Saura, Gonzalo de la Morena, María D. Espinosa
Publikováno v:
Journal of the American Society of Echocardiography. 30:28-35
Background Left ventricular outflow tract (LVOT) measurement is a critical step in the quantification of aortic valve area. The assumption of a circular morphology of the LVOT may induce some errors. The aim of this study was to assess the three-dime
Publikováno v:
ICCS
Based on a software tool proposed for the automatic quantification of the exact hyper- trabeculation degree in the left ventricle myocardium, where the trabecular and the compacted zones are detected, we have designed, developed and tested a training