Zobrazeno 1 - 10
of 34
pro vyhledávání: '"Josan Chung"'
Publikováno v:
PLoS Pathogens, Vol 13, Iss 5, p e1006416 (2017)
[This corrects the article DOI: 10.1371/journal.ppat.1003490.].
Externí odkaz:
https://doaj.org/article/be99bd7c59ee485d870305075d82aed9
Publikováno v:
PLoS Pathogens, Vol 12, Iss 12, p e1006086 (2016)
[This corrects the article DOI: 10.1371/journal.ppat.1003490.].
Externí odkaz:
https://doaj.org/article/cf33e0584eae4d5ea3c753ca452c5d62
Publikováno v:
PLoS Pathogens, Vol 9, Iss 7, p e1003490 (2013)
The intrahepatic immune environment is normally biased towards tolerance. Nonetheless, effective antiviral immune responses can be induced against hepatotropic pathogens. To examine the immunological basis of this paradox we studied the ability of he
Externí odkaz:
https://doaj.org/article/57ac6d0558bd40f5a4bdcafb9712e526
Autor:
Christina Whitten-Bauer, Michael D. Huber, Larry Gerace, Urtzi Garaigorta, Andoni Gómez-Moreno, Pilar Gomollón-Zueco, Josan Chung
Publikováno v:
Cells
Volume 8
Issue 12
Digital.CSIC. Repositorio Institucional del CSIC
instname
Volume 8
Issue 12
Digital.CSIC. Repositorio Institucional del CSIC
instname
© 2019 by the authors.
Development of hepatitis C virus (HCV) infection cell culture systems has permitted the identification of cellular factors that regulate the HCV life cycle. Some of these cellular factors affect steps in the viral life cy
Development of hepatitis C virus (HCV) infection cell culture systems has permitted the identification of cellular factors that regulate the HCV life cycle. Some of these cellular factors affect steps in the viral life cy
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f91d49de40ba475d0d8c4d4dcda0d6db
http://hdl.handle.net/10261/197155
http://hdl.handle.net/10261/197155
Autor:
Diego Calabrese, Josan Chung, Markus H. Heim, Benedetta Campana, Zuzanna Makowska, Francis V. Chisari, Stefan Wieland, Michael T. Dill
Publikováno v:
Hepatology (Baltimore Md.)
Background and Rationale: Approximately 50% of patients with chronic hepatitis C (CHC) have ongoing expression of interferon stimulated genes (ISGs) in the liver. It is unclear why this endogenous antiviral response is inefficient in eradicating the
Autor:
Holly Maier, Alana Althage, Stefan Wieland, Francis V. Chisari, Josan Chung, Masanori Isogawa, Priscilla L. Yang
Publikováno v:
Proceedings of the National Academy of Sciences. 107:798-802
To better define the mechanism(s) likely responsible for viral clearance during hepatitis B virus (HBV) infection, viral clearance was studied in a panel of immunodeficient mouse strains that were hydrodynamically transfected with a plasmid containin
Autor:
Jane A. McKeating, Zania Stamataki, Francis V. Chisari, Guofeng Cheng, Pablo Gastaminza, Jin Zhong, Masanori Isogawa, Josan Chung
Publikováno v:
Journal of Virology
The virological and cellular consequences of persistent hepatitis C virus (HCV) infection have been elusive due to the absence of the requisite experimental systems. Here, we report the establishment and the characteristics of persistent in vitro inf
Autor:
Francis V. Chisari, Christopher M. Walker, John Sidney, Shinichi Asabe, Josan Chung, Alessandro Sette, Bjoern Peters, Robert H. Purcell, Kelly-Anne Purton, Timothy J. Pencille
Publikováno v:
Publons
The chimpanzee (Pan troglodytes) is an important model for studying the immune response to several human pathogens, but the study of correlates of immunity has been hindered by the fact that little is known about the epitope-binding specificity of ch
Publikováno v:
Proceedings of the National Academy of Sciences. 99:13825-13830
Hepatitis B virus (HBV) is a prototype for liver-specific pathogens in which the failure of the immune system to mount an effective response leads to chronic infection. Our understanding of the immune response to HBV is incomplete, largely due to the
Publikováno v:
Journal of Virology. 76:8609-8620
Persistent hepatitis B virus (HBV) infection is characterized by a weak and narrowly focused CD8+T-cell response to HBV that is thought to reflect the induction of central and/or peripheral tolerance to HBV proteins in neonatal and adult onset infect