Zobrazeno 1 - 4
of 4
pro vyhledávání: '"José Gerardo Teles Reis"'
Publikováno v:
International Journal of Molecular Sciences, Vol 22, Iss 16, p 8873 (2021)
The phenomenon of how oncogenes and tumor-suppressor mutations can synergize to promote tumor fitness and cancer progression can be studied in relatively simple animal model systems such as Drosophila melanogaster. Almost two decades after the landma
Externí odkaz:
https://doaj.org/article/dec4f8ba8bda4c6c8613d58e755fb083
Autor:
Petter Holland, Todd Andrew Schoborg, Ifat Abramovich, Szabolcs Takáts, Caroline Dillard, Ashish Jain, Fergal O'Farrell, Sebastian Wolfgang Schultz, William M. Hagopian, Eduardo Martin Quintana, Rachel Ng, Nadja Sandra Katheder, Mohammed Mahidur Rahman, José Gerardo Teles Reis, Andreas Brech, Heinrich Jasper, Nasser M. Rusan, Anne Hope Jahren, Eyal Gottlieb, Tor Erik Rusten
Publikováno v:
Molecular Cancer Research. 21:IA14-IA14
During tumor growth-when nutrient and anabolic demands are high-autophagy supports tumor metabolism and growth through lysosomal organelle turnover and nutrient recycling. Ras-driven tumors additionally invoke non-autonomous autophagy in the microenv
Publikováno v:
International Journal of Molecular Sciences
International Journal of Molecular Sciences, Vol 22, Iss 8873, p 8873 (2021)
International Journal of Molecular Sciences, Vol 22, Iss 8873, p 8873 (2021)
The phenomenon of how oncogenes and tumor-suppressor mutations can synergize to promote tumor fitness and cancer progression can be studied in relatively simple animal model systems such as Drosophila melanogaster. Almost two decades after the landma
Autor:
Todd A. Schoborg, Eyal Gottlieb, Rachel K. Ng, Nadja Sandra Katheder, Nasser M. Rusan, Mohammed Mahidur Rahman, William M. Hagopian, Ashish Jain, José Gerardo Teles Reis, Szabolcs Takats, Tor Erik Rusten, Petter Holland, Ifat Abramovich, Rojyar Khezri, Caroline Dillard, Anne Hope Jahren, Eduardo Martin Quintana, Andreas Brech, Sebastian W. Schultz, Heinrich Jasper, Fergal O'Farrell
Publikováno v:
The EMBO Journal
During tumor growth—when nutrient and anabolic demands are high—autophagy supports tumor metabolism and growth through lysosomal organelle turnover and nutrient recycling. Ras‐driven tumors additionally invoke non‐autonomous autophagy in the