Zobrazeno 1 - 8
of 8
pro vyhledávání: '"Jonny Daborg"'
Autor:
Ulf Andreasson, Jonny Daborg, Henrik Zetterberg, Eric Hanse, Andreas Bjorefeldt, Ilse Riebe, Pontus Wasling
Publikováno v:
The Journal of Physiology. 593:231-243
The cerebrospinal fluid contains numerous neuromodulators at ambient levels but whether, and how, they affect the activity of central neurons is unknown. This study provides experimental evidence that human cerebrospinal fluid (hCSF) increases the ex
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::626699473dad94e599374de6518f9d35
https://doi.org/10.1113/jphysiol.2014.284711
https://doi.org/10.1113/jphysiol.2014.284711
Autor:
Henrik Zetterberg, Anna Stokowska, Marie Kalm, Christine T. Ekdahl, Klas Blomgren, Pontus Wasling, Karl E. Carlström, Marta Perez-Alcazar, Eric Hanse, Marcela Pekna, Andreas Bjorefeldt, Jonny Daborg
Publikováno v:
Experimental Neurology. 253:154-164
Previous work implicated the complement system in adult neurogenesis as well as elimination of synapses in the developing and injured CNS. In the present study, we used mice lacking the third complement component (C3) to elucidate the role the comple
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::29909ce7c0fef978b756f6721c68e134
Autor:
Eric Hanse, Jonny Daborg, Sandra Holmgren, Marcela Pekna, Ulf Andreasson, Staffan Nilsson, Kaj Blennow, Henrik Zetterberg, Ingmar Skoog, Alexandra Abramsson, Lennart Minthon, Madeleine Zetterberg
Publikováno v:
Journal of Alzheimer's Disease. 35:51-57
The complement system has been implicated in both physiological synapse elimination and Alzheimer's disease (AD). Here, we investigated associations between four single nucleotide polymorphisms (SNPs) in complement genes and cerebrospinal fluid (CSF)
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::979350d34198d5dfbdd81258ee774e31
https://doi.org/10.3233/jad-121930
https://doi.org/10.3233/jad-121930
Autor:
Malin von Otter, Jonny Daborg, Henrik Zetterberg, Kaj Blennow, Annica Sjölander, Lennart Minthon, Staffan Nilsson, Deborah Gustafson, Ingmar Skoog
Publikováno v:
Journal of Neural Transmission
The receptor for advanced glycation end-products (RAGE) has been implicated in several pathophysiological processes relevant to Alzheimer's disease (AD), including transport and synaptotoxicity of AD-associated amyloid beta (A beta) peptides. A recen
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::f78b7316063270a68ef845dbcfb58e17
http://europepmc.org/articles/PMC2895876
http://europepmc.org/articles/PMC2895876
Autor:
Jonny Daborg, Lennart Minthon, Henrik Zetterberg, Eric Hanse, Ulf Andreasson, Marcela Pekna, Ronald Lautner, Oskar Hansson, Kaj Blennow
Publikováno v:
Journal of neural transmission (Vienna, Austria : 1996). 119(7)
Alzheimer’s disease (AD) is strongly associated with loss of synapses. The complement system has been shown to be involved in synaptic elimination. Several studies point to an association between AD and the complement system. The purpose of this st
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::41a295e3a9d76e390ca1318048db6dd7
https://pubmed.ncbi.nlm.nih.gov/22488444
https://pubmed.ncbi.nlm.nih.gov/22488444
Autor:
Ingmar Skoog, Henrik Zetterberg, Eric Hanse, Deborah Gustafson, Malin von Otter, Lennart Minthon, Annica Sjölander, Kaj Blennow, Jonny Daborg
Publikováno v:
Alzheimer's & Dementia. 6
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_________::7686493b1510fee645117914f1e401da
https://doi.org/10.1016/j.jalz.2010.08.077
https://doi.org/10.1016/j.jalz.2010.08.077
Autor:
Jonny Daborg, Harald Hampel, Mikael K. Gustavsson, Henrik Zetterberg, Kaj Blennow, Oskar Hansson, Ulf Andreasson, Douglas Galasko, Eric Hanse, Peder Buchhave, Katharina Buerger, John M. Ringman, Andreas Harmsen, Erik Portelius
Publikováno v:
Molecular Neurodegeneration, Vol 5, Iss 1, p 2 (2010)
Molecular Neurodegeneration
Molecular Neurodegeneration
Background Alzheimer's disease (AD) is associated with deposition of amyloid β (Aβ) in the brain, which is reflected by low concentration of the Aβ1-42 peptide in the cerebrospinal fluid (CSF). There are at least 15 additional Aβ peptides in huma
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::b637b59088fb1575e0d77656e3547f2c
https://doi.org/10.1186/1750-1326-5-2
https://doi.org/10.1186/1750-1326-5-2
Autor:
Eric Hanse, Ilse Riebe, My Andersson, Henrik Zetterberg, Pontus Wasling, Jonny Daborg, Erik Portelius, Kaj Blennow
Publikováno v:
Journal of Alzheimer's disease : JAD. 16(1)
Pathological hallmarks of Alzheimer's disease (AD) include synaptic and neuronal degeneration and the presence of extracellular deposits of amyloid-beta (Abeta) in senile plaques in the cerebral cortex. Although these brain lesions may be seen also i
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::85957114e914a73c84cb970e9f7e1a65
https://pubmed.ncbi.nlm.nih.gov/19158416
https://pubmed.ncbi.nlm.nih.gov/19158416