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pro vyhledávání: '"Jonish-Grossman A"'
Autor:
Raoul Orvieto, Anat Jonish-Grossman, Sharon Avhar Maydan, Meirav Noach-Hirsh, Olga Dratviman-Storobinsky, Adva Aizer
Publikováno v:
Reproductive Biology and Endocrinology, Vol 20, Iss 1, Pp 1-5 (2022)
Summary Embryo transfer is a crucial step in IVF cycle, with increasing trend during the last decade of transferring a single embryo, preferably at the blastocyst stage. Despite increasing evidence supporting Day 5 blastocyst-stage transfer, the opti
Externí odkaz:
https://doaj.org/article/0f80d5e60fc44460952c2b552db6a31b
Publikováno v:
Reproductive Biology and Endocrinology, Vol 18, Iss 1, Pp 1-6 (2020)
Abstract Human embryogenesis frequently coinciding with cell division mistakes contributing to pervasive embryonic aneuploidy/mosaicism. While embryo self-correction was elegantly demonstrated in mouse models, human studies are lacking. Here we are w
Externí odkaz:
https://doaj.org/article/ebe1e21164ec40689d932f168eeca9c6
Autor:
Orvieto, Raoul1,2,3 (AUTHOR) raoul.orvieto@sheba.health.gov.il, Jonish-Grossman, Anat4 (AUTHOR), Maydan, Sharon Avhar1,2 (AUTHOR), Noach-Hirsh, Meirav1 (AUTHOR), Dratviman-Storobinsky, Olga1 (AUTHOR), Aizer, Adva1,2 (AUTHOR)
Publikováno v:
Reproductive Biology & Endocrinology. 3/17/2022, Vol. 20 Issue 1, p1-5. 5p.
Publikováno v:
Reproductive Biology and Endocrinology, Vol 18, Iss 1, Pp 1-6 (2020)
Reproductive Biology and Endocrinology : RB&E
Reproductive Biology and Endocrinology : RB&E
Human embryogenesis frequently coinciding with cell division mistakes contributing to pervasive embryonic aneuploidy/mosaicism. While embryo self-correction was elegantly demonstrated in mouse models, human studies are lacking. Here we are witness to
Autor:
Orvieto, Raoul, Shimon, Chen, Rienstein, Shlomit, Jonish-Grossman, Anat, Shani, Hagit, Adva Aizer
Additional file 1. Time-lapse EmbryoScope™ photography of embryo expelling cell debris/cell fragments within the zona pellucida ( https://youtu.be/3RNUJ4iW0IE ).
Externí odkaz:
https://explore.openaire.eu/search/publication?articleId=doi_dedup___::13348074f9b5a15bbff29c1bf69388c8
Akademický článek
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Publikováno v:
Human Reproduction. 22:2538-2545
BACKGROUND The pathologic features of Down syndrome are assumed to be the result of over-expression of genes located on chromosome 21 and/or a more global transcriptional misregulation that crosses chromosomal borders. METHODS To address this issue,
Akademický článek
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